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Crkl deficiency disrupts Fgf8 signaling in a mouse model of 22q11 deletion syndromes.
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Crkl deficiency disrupts Fgf8 signaling in a mouse model of 22q11 deletion syndromes.
Crkl deficiency disrupts Fgf8 signaling in a mouse model of 22q11 deletion syndromes. Dev Cell. 2006 Jan; 10(1):71-80.
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PubMed
subject areas
Animals
Apoptosis
Blotting, Western
Bone and Bones
Cardiovascular System
Cell Count
Cells, Cultured
Chemotactic Factors
Chromosomes, Human, Pair 22
DiGeorge Syndrome
Disease Models, Animal
Embryo, Mammalian
Enzyme Activation
Fibroblast Growth Factor 8
Fluorescent Antibody Technique
Gene Deletion
Gene Expression Regulation, Developmental
Genotype
Humans
Mice
Mice, Knockout
Models, Biological
Neural Crest
Pharynx
Phenotype
Proto-Oncogene Proteins c-crk
Receptors, Fibroblast Growth Factor
Signal Transduction
Time Factors
authors with profiles
Akira Imamoto