"Myocytes, Cardiac" is a descriptor in the National Library of Medicine's controlled vocabulary thesaurus,
MeSH (Medical Subject Headings). Descriptors are arranged in a hierarchical structure,
which enables searching at various levels of specificity.
Striated muscle cells found in the heart. They are derived from cardiac myoblasts (MYOBLASTS, CARDIAC).
| Descriptor ID |
D032383
|
| MeSH Number(s) |
A07.541.704.570 A10.690.552.750.570 A11.620.500
|
| Concept/Terms |
Myocytes, Cardiac- Myocytes, Cardiac
- Cardiac Myocyte
- Cardiac Myocytes
- Myocyte, Cardiac
- Muscle Cells, Heart
- Cell, Heart Muscle
- Cells, Heart Muscle
- Heart Muscle Cell
- Heart Muscle Cells
- Muscle Cell, Heart
- Cardiomyocytes
- Cardiomyocyte
- Muscle Cells, Cardiac
- Cardiac Muscle Cell
- Cardiac Muscle Cells
- Cell, Cardiac Muscle
- Cells, Cardiac Muscle
- Muscle Cell, Cardiac
|
Below are MeSH descriptors whose meaning is more general than "Myocytes, Cardiac".
Below are MeSH descriptors whose meaning is more specific than "Myocytes, Cardiac".
This graph shows the total number of publications written about "Myocytes, Cardiac" by people in this website by year, and whether "Myocytes, Cardiac" was a major or minor topic of these publications.
To see the data from this visualization as text,
click here.
| Year | Major Topic | Minor Topic | Total |
|---|
| 2002 | 1 | 4 | 5 |
| 2003 | 8 | 6 | 14 |
| 2004 | 6 | 6 | 12 |
| 2005 | 1 | 4 | 5 |
| 2006 | 8 | 0 | 8 |
| 2007 | 5 | 3 | 8 |
| 2008 | 2 | 4 | 6 |
| 2009 | 6 | 4 | 10 |
| 2010 | 11 | 5 | 16 |
| 2011 | 6 | 4 | 10 |
| 2012 | 10 | 4 | 14 |
| 2013 | 4 | 4 | 8 |
| 2014 | 16 | 2 | 18 |
| 2015 | 7 | 3 | 10 |
| 2016 | 13 | 1 | 14 |
| 2017 | 9 | 2 | 11 |
| 2018 | 8 | 2 | 10 |
| 2019 | 6 | 8 | 14 |
| 2020 | 6 | 2 | 8 |
| 2021 | 8 | 1 | 9 |
| 2022 | 4 | 3 | 7 |
| 2023 | 1 | 3 | 4 |
| 2024 | 5 | 5 | 10 |
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Below are the most recent publications written about "Myocytes, Cardiac" by people in Profiles.
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Endothelial oestrogen-myocardial cyclic guanosine monophosphate axis critically determines angiogenesis and cardiac performance during pressure overload. Cardiovasc Res. 2024 Dec 04; 120(15):1884-1897.
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Simultaneous electromechanical monitoring in engineered heart tissues using a mesoscale framework. Sci Adv. 2024 Sep 13; 10(37):eado7089.
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Proteolytic degradation of atrial sarcomere proteins underlies contractile defects in atrial fibrillation. Am J Physiol Heart Circ Physiol. 2024 08 01; 327(2):H460-H472.
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Pervasive nuclear envelope ruptures precede ECM signaling and disease onset without activating cGAS-STING in Lamin-cardiomyopathy mice. Cell Rep. 2024 06 25; 43(6):114284.
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Reduction of Filamin C Results in Altered Proteostasis, Cardiomyopathy, and Arrhythmias. J Am Heart Assoc. 2024 May 21; 13(10):e030467.
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Susceptibility to innate immune activation in genetically mediated myocarditis. J Clin Invest. 2024 May 16; 134(13).
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In Vivo Mapping of Myocardial Injury Outside the Infarct Zone: Tissue at an Intermediate Pathological State. J Am Heart Assoc. 2024 May 07; 13(9):e032577.
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Monolithic silicon for high spatiotemporal translational photostimulation. Nature. 2024 Feb; 626(8001):990-998.
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Physiological stress improves stem cell modeling of dystrophic cardiomyopathy. Dis Model Mech. 2024 Jun 01; 17(6).
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Dynamin-related protein 1 is a critical regulator of mitochondrial calcium homeostasis during myocardial ischemia/reperfusion injury. FASEB J. 2024 01; 38(1):e23379.