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Connection

Yoav Gilad to Myocytes, Cardiac

This is a "connection" page, showing publications Yoav Gilad has written about Myocytes, Cardiac.
Connection Strength

3.648
  1. Dynamic effects of genetic variation on gene expression revealed following hypoxic stress in cardiomyocytes. Elife. 2021 02 08; 10.
    View in: PubMed
    Score: 0.607
  2. Dynamic genetic regulation of gene expression during cellular differentiation. Science. 2019 06 28; 364(6447):1287-1290.
    View in: PubMed
    Score: 0.543
  3. A generally conserved response to hypoxia in iPSC-derived cardiomyocytes from humans and chimpanzees. Elife. 2019 04 08; 8.
    View in: PubMed
    Score: 0.534
  4. A Comparative Assessment of Human and Chimpanzee iPSC-derived Cardiomyocytes with Primary Heart Tissues. Sci Rep. 2018 10 17; 8(1):15312.
    View in: PubMed
    Score: 0.517
  5. Determining the genetic basis of anthracycline-cardiotoxicity by molecular response QTL mapping in induced cardiomyocytes. Elife. 2018 05 08; 7.
    View in: PubMed
    Score: 0.502
  6. Impact of regulatory variation across human iPSCs and differentiated cells. Genome Res. 2018 01; 28(1):122-131.
    View in: PubMed
    Score: 0.487
  7. Single-cell sequencing reveals lineage-specific dynamic genetic regulation of gene expression during human cardiomyocyte differentiation. PLoS Genet. 2022 01; 18(1):e1009666.
    View in: PubMed
    Score: 0.162
  8. Functional dynamic genetic effects on gene regulation are specific to particular cell types and environmental conditions. Elife. 2021 05 14; 10.
    View in: PubMed
    Score: 0.155
  9. Systematic Comparison of High-throughput Single-Cell and Single-Nucleus Transcriptomes during Cardiomyocyte Differentiation. Sci Rep. 2020 01 30; 10(1):1535.
    View in: PubMed
    Score: 0.141
Connection Strength

The connection strength for concepts is the sum of the scores for each matching publication.

Publication scores are based on many factors, including how long ago they were written and whether the person is a first or senior author.