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Connection

Raghu Mirmira to Mice, Inbred C57BL

This is a "connection" page, showing publications Raghu Mirmira has written about Mice, Inbred C57BL.
Connection Strength

0.804
  1. Phenotypic sexual dimorphism in response to dietary fat manipulation in C57BL/6J mice. J Diabetes Complications. 2021 02; 35(2):107795.
    View in: PubMed
    Score: 0.113
  2. Hypusinated eIF5A is expressed in the pancreas and spleen of individuals with type 1 and type 2 diabetes. PLoS One. 2020; 15(3):e0230627.
    View in: PubMed
    Score: 0.107
  3. Maintenance of Pdx1 mRNA translation in islet ß-cells during the unfolded protein response. Mol Endocrinol. 2014 Nov; 28(11):1820-30.
    View in: PubMed
    Score: 0.073
  4. Protective effects of polyamine depletion in mouse models of type 1 diabetes: implications for therapy. Amino Acids. 2014 Mar; 46(3):633-42.
    View in: PubMed
    Score: 0.067
  5. Mouse islet of Langerhans isolation using a combination of purified collagenase and neutral protease. J Vis Exp. 2012 Sep 07; (67).
    View in: PubMed
    Score: 0.064
  6. The unique hypusine modification of eIF5A promotes islet beta cell inflammation and dysfunction in mice. J Clin Invest. 2010 Jun; 120(6):2156-70.
    View in: PubMed
    Score: 0.054
  7. Noninvasive assessment of pancreatic beta-cell function in vivo with manganese-enhanced magnetic resonance imaging. Am J Physiol Endocrinol Metab. 2009 Mar; 296(3):E573-8.
    View in: PubMed
    Score: 0.049
  8. Methyltransferase Set7/9 maintains transcription and euchromatin structure at islet-enriched genes. Diabetes. 2009 Jan; 58(1):185-93.
    View in: PubMed
    Score: 0.049
  9. Oligomeric collagen as an encapsulation material for islet/ß-cell replacement: effect of islet source, dose, implant site, and administration format. Am J Physiol Endocrinol Metab. 2020 08 01; 319(2):E388-E400.
    View in: PubMed
    Score: 0.027
  10. Hypusine biosynthesis in ß cells links polyamine metabolism to facultative cellular proliferation to maintain glucose homeostasis. Sci Signal. 2019 12 03; 12(610).
    View in: PubMed
    Score: 0.026
  11. Interleukin-6 Reduces ß-Cell Oxidative Stress by Linking Autophagy With the Antioxidant Response. Diabetes. 2018 08; 67(8):1576-1588.
    View in: PubMed
    Score: 0.024
  12. Loss of Free Fatty Acid Receptor 2 leads to impaired islet mass and beta cell survival. Sci Rep. 2016 06 21; 6:28159.
    View in: PubMed
    Score: 0.021
  13. An Acetate-Specific GPCR, FFAR2, Regulates Insulin Secretion. Mol Endocrinol. 2015 Jul; 29(7):1055-66.
    View in: PubMed
    Score: 0.019
  14. Transcriptional activity of the islet ß cell factor Pdx1 is augmented by lysine methylation catalyzed by the methyltransferase Set7/9. J Biol Chem. 2015 Apr 10; 290(15):9812-22.
    View in: PubMed
    Score: 0.019
  15. Insulin regulates carboxypeptidase E by modulating translation initiation scaffolding protein eIF4G1 in pancreatic ß cells. Proc Natl Acad Sci U S A. 2014 Jun 03; 111(22):E2319-28.
    View in: PubMed
    Score: 0.018
  16. Divergent compensatory responses to high-fat diet between C57BL6/J and C57BLKS/J inbred mouse strains. Am J Physiol Endocrinol Metab. 2013 Dec; 305(12):E1495-511.
    View in: PubMed
    Score: 0.017
  17. Mitogen-inducible gene 6 triggers apoptosis and exacerbates ER stress-induced ß-cell death. Mol Endocrinol. 2013 Jan; 27(1):162-71.
    View in: PubMed
    Score: 0.016
  18. An intracellular role for ABCG1-mediated cholesterol transport in the regulated secretory pathway of mouse pancreatic beta cells. J Clin Invest. 2010 Jul; 120(7):2575-89.
    View in: PubMed
    Score: 0.014
  19. The roles of ATF3, an adaptive-response gene, in high-fat-diet-induced diabetes and pancreatic beta-cell dysfunction. Mol Endocrinol. 2010 Jul; 24(7):1423-33.
    View in: PubMed
    Score: 0.014
  20. AGI-1067, a novel antioxidant and anti-inflammatory agent, enhances insulin release and protects mouse islets. Mol Cell Endocrinol. 2010 Jul 29; 323(2):246-55.
    View in: PubMed
    Score: 0.013
Connection Strength

The connection strength for concepts is the sum of the scores for each matching publication.

Publication scores are based on many factors, including how long ago they were written and whether the person is a first or senior author.