D018374AnatomyA11.251.210.520A11.436.520110.999955LLC-PK1 Cellsprns:emailEncryptedemail addressprns:fullNamefull nameprns:hasAuthorListauthor listprns:hasNetworkhas networkprns:hasPublicationVenuepublished inprns:informationResourceReferenceinformation resource referenceprns:isPrimaryPositionis primary positionprns:latitudelatitudeprns:longitudelongitudeprns:mainImagephotoprns:maxWeightmaximum weightprns:medlineTAjournal title abbreviationprns:meshDescriptorUIMeSH DescriptorUIprns:meshSemanticGroupNameMeSH semantic group nameprns:meshTreeNumberMeSH tree numberprns:minWeightminimum weightprns:numberOfAuthorsnumber of authorsprns:numberOfConnectionsnumber of connectionsprns:numberOfPublicationsnumber of publicationsprns:personIdPerson IDprns:personInPrimaryPositionperson in primary positionprns:positionInDepartmentposition in departmentprns:predicateNodepredicate nodeprns:publicationDatepublication dateprns:sortOrdersort orderprns:uniquenessWeightuniqueness weightprns:yearyearAcademic ArticleArticleDocumentbibo:pmidPubMed IdentifierDepartmentvivo:hrJobTitleHR job titleInformation ResourcePositionvivo:positionInOrganizationposition in organizationvivo:preferredTitlepreferred titlevivo:researchAreaOfresearch area ofvivo:subjectAreaForsubject area forrdf:predicatepredicaterdf:typetyperdfs:labellabelConceptAgentfoaf:firstNamefirst namefoaf:lastNamelast nameOrganizationPersonMedicine-Gastroenterology8648261Soleimani M, Singh G, Bookstein C, Rao MC, Chang EB, Dominguez JHThe Journal of laboratory and clinical medicineInhibition of glycosylation decreases Na+/H+ exchange activity, blocks NHE-3 transport to the membrane, and increases NHE-3 mRNA expression in LLC-PK1 cells. J Lab Clin Med. 1996 Jun; 127(6):565-73.J Lab Clin Med1996-06-01T00:00:001996Inhibition of glycosylation decreases Na+/H+ exchange activity, blocks NHE-3 transport to the membrane, and increases NHE-3 mRNA expression in LLC-PK1 cells.0.009085180.009085181research area of0.06183930.03425955subject area forUniversity of ChicagoEugene B.Chang0mBqA6AbHszMknJtY0fp/Cv/+94=Eugene B. Chang41.78927490000000-87.601250000000001255Chang, Eugene B.Professortrue1ProfessorProfessor7626087Nabhan C, Xiong Y, Xie LY, Abou-Samra ABBiochemical and biophysical research communicationsThe alternatively spliced type II corticotropin-releasing factor receptor, stably expressed in LLCPK-1 cells, is not well coupled to the G protein(s). Biochem Biophys Res Commun. 1995 Jul 26; 212(3):1015-21.Biochem Biophys Res Commun1995-07-26T00:00:001995The alternatively spliced type II corticotropin-releasing factor receptor, stably expressed in LLCPK-1 cells, is not well coupled to the G protein(s).9362333Becker BN, Cheng HF, Harris RCThe American journal of physiologyApical ANG II-stimulated PLA2 activity and Na+ flux: a potential role for Ca2+-independent PLA2. Am J Physiol. 1997 10; 273(4):F554-62.Am J Physiol1997-10-01T00:00:001997Apical ANG II-stimulated PLA2 activity and Na+ flux: a potential role for Ca2+-independent PLA2.14742678Becker BN, Cheng HF, Hammond TG, Harris RCMolecular pharmacologyThe type 1 angiotensin II receptor tail affects receptor targeting, internalization, and membrane fusion properties. Mol Pharmacol. 2004 Feb; 65(2):362-9.Mol Pharmacol2004-02-01T00:00:002004The type 1 angiotensin II receptor tail affects receptor targeting, internalization, and membrane fusion properties.10409234Jilling T, Haddad IY, Cheng SH, Matalon SThe American journal of physiologyNitric oxide inhibits heterologous CFTR expression in polarized epithelial cells. Am J Physiol. 1999 07; 277(1):L89-96.Am J Physiol1999-07-01T00:00:001999Nitric oxide inhibits heterologous CFTR expression in polarized epithelial cells.