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Connection

Co-Authors

This is a "connection" page, showing publications co-authored by Emily Rogalski and Adam Martersteck.
Connection Strength

4.251
  1. Focal amyloid and asymmetric tau in an imaging-to-autopsy case of clinical primary progressive aphasia with Alzheimer disease neuropathology. Acta Neuropathol Commun. 2022 08 09; 10(1):111.
    View in: PubMed
    Score: 0.756
  2. Relationships among tau burden, atrophy, age, and naming in the aphasic variant of Alzheimer's disease. Alzheimers Dement. 2021 11; 17(11):1788-1797.
    View in: PubMed
    Score: 0.709
  3. Differential neurocognitive network perturbation in amnestic and aphasic Alzheimer disease. Neurology. 2020 02 18; 94(7):e699-e704.
    View in: PubMed
    Score: 0.634
  4. The causal association between resting state intrinsic functional networks and neurodegeneration. Brain Commun. 2025; 7(2):fcaf098.
    View in: PubMed
    Score: 0.226
  5. Clinical Manifestations. Alzheimers Dement. 2024 Dec; 20 Suppl 3:e090906.
    View in: PubMed
    Score: 0.222
  6. SuperAging functional connectomics from resting-state functional MRI. Brain Commun. 2024; 6(4):fcae205.
    View in: PubMed
    Score: 0.215
  7. Modularity and granularity across the language network-A primary progressive aphasia perspective. Cortex. 2021 08; 141:482-496.
    View in: PubMed
    Score: 0.174
  8. Functional decline in the aphasic variant of Alzheimer's disease. Alzheimers Dement. 2021 10; 17(10):1641-1648.
    View in: PubMed
    Score: 0.172
  9. Clinical and cortical decline in the aphasic variant of Alzheimer's disease. Alzheimers Dement. 2019 04; 15(4):543-552.
    View in: PubMed
    Score: 0.148
  10. Proof of concept demonstration of optimal composite MRI endpoints for clinical trials. Alzheimers Dement (N Y). 2016 Sep; 2(3):177-181.
    View in: PubMed
    Score: 0.125
  11. Aphasic variant of Alzheimer disease: Clinical, anatomic, and genetic features. Neurology. 2016 Sep 27; 87(13):1337-43.
    View in: PubMed
    Score: 0.125
  12. Is in vivo amyloid distribution asymmetric in primary progressive aphasia? Ann Neurol. 2016 Mar; 79(3):496-501.
    View in: PubMed
    Score: 0.120
  13. Asymmetry of cortical decline in subtypes of primary progressive aphasia. Neurology. 2014 Sep 23; 83(13):1184-91.
    View in: PubMed
    Score: 0.109
  14. Longitudinal neuropsychological performance of cognitive SuperAgers. J Am Geriatr Soc. 2014 Aug; 62(8):1598-600.
    View in: PubMed
    Score: 0.108
  15. Naming vs knowing faces in primary progressive aphasia: a tale of 2 hemispheres. Neurology. 2013 Aug 13; 81(7):658-64.
    View in: PubMed
    Score: 0.101
  16. A novel frontal pathway underlies verbal fluency in primary progressive aphasia. Brain. 2013 Aug; 136(Pt 8):2619-28.
    View in: PubMed
    Score: 0.101
  17. Democratizing dementia research through global online conferences. Nat Neurosci. 2025 Jun; 28(6):1109-1112.
    View in: PubMed
    Score: 0.057
  18. Geometric deep learning reveals a structuro-temporal understanding of healthy and pathologic brain aging. Front Aging Neurosci. 2022; 14:895535.
    View in: PubMed
    Score: 0.047
  19. Accumulation of neurofibrillary tangles and activated microglia is associated with lower neuron densities in the aphasic variant of Alzheimer's disease. Brain Pathol. 2021 01; 31(1):189-204.
    View in: PubMed
    Score: 0.042
  20. Frontotemporal networks and behavioral symptoms in primary progressive aphasia. Neurology. 2016 04 12; 86(15):1393-1399.
    View in: PubMed
    Score: 0.030
  21. Morphometric and histologic substrates of cingulate integrity in elders with exceptional memory capacity. J Neurosci. 2015 Jan 28; 35(4):1781-91.
    View in: PubMed
    Score: 0.028
Connection Strength

The connection strength for concepts is the sum of the scores for each matching publication.

Publication scores are based on many factors, including how long ago they were written and whether the person is a first or senior author.