Eugene B. Chang to Sepsis
This is a "connection" page, showing publications Eugene B. Chang has written about Sepsis.
Connection Strength
0.760
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The re-emerging role of the intestinal microflora in critical illness and inflammation: why the gut hypothesis of sepsis syndrome will not go away. J Leukoc Biol. 2008 Mar; 83(3):461-6.
Score: 0.240
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Exosomes from Endothelial Progenitor Cells Improve the Outcome of a Murine Model of Sepsis. Mol Ther. 2018 05 02; 26(5):1375-1384.
Score: 0.122
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Endothelial progenitor cells and a stromal cell-derived factor-1a analogue synergistically improve survival in sepsis. Am J Respir Crit Care Med. 2014 Jun 15; 189(12):1509-19.
Score: 0.094
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Oral PEG 15-20 protects the intestine against radiation: role of lipid rafts. Am J Physiol Gastrointest Liver Physiol. 2009 Dec; 297(6):G1041-52.
Score: 0.068
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Common problems in critically ill obstetric patients, with an emphasis on pharmacotherapy. Am J Med Sci. 2008 Jan; 335(1):65-70.
Score: 0.060
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Chronic acid water feeding protects mice against lethal gut-derived sepsis due to Pseudomonas aeruginosa. Curr Issues Intest Microbiol. 2006 Mar; 7(1):19-28.
Score: 0.053
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Glutamine reduces cytokine release, organ damage, and mortality in a rat model of endotoxemia. Shock. 2001 Nov; 16(5):398-402.
Score: 0.039
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Gut-derived sepsis occurs when the right pathogen with the right virulence genes meets the right host: evidence for in vivo virulence expression in Pseudomonas aeruginosa. Ann Surg. 2000 Oct; 232(4):480-9.
Score: 0.036
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The key role of Pseudomonas aeruginosa PA-I lectin on experimental gut-derived sepsis. Ann Surg. 2000 Jul; 232(1):133-42.
Score: 0.036
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Pseudomonas aeruginosa expresses a lethal virulence determinant, the PA-I lectin/adhesin, in the intestinal tract of a stressed host: the role of epithelia cell contact and molecules of the Quorum Sensing Signaling System. Ann Surg. 2003 Nov; 238(5):754-64.
Score: 0.011