The University of Chicago Header Logo

Connection

Nanduri R. Prabhakar to Rats

This is a "connection" page, showing publications Nanduri R. Prabhakar has written about Rats.
Connection Strength

3.596
  1. Lysine demethylase KDM6B regulates HIF-1a-mediated systemic and cellular responses to intermittent hypoxia. Physiol Genomics. 2021 09 01; 53(9):385-394.
    View in: PubMed
    Score: 0.123
  2. Hypoxia-inducible factors and obstructive sleep apnea. J Clin Invest. 2020 10 01; 130(10):5042-5051.
    View in: PubMed
    Score: 0.116
  3. Long-term facilitation of catecholamine secretion from adrenal chromaffin cells of neonatal rats by chronic intermittent hypoxia. J Neurophysiol. 2019 11 01; 122(5):1874-1883.
    View in: PubMed
    Score: 0.108
  4. H2S mediates carotid body response to hypoxia but not anoxia. Respir Physiol Neurobiol. 2019 01; 259:75-85.
    View in: PubMed
    Score: 0.100
  5. Measurement of Sensory Nerve Activity from the Carotid Body. Methods Mol Biol. 2018; 1742:115-124.
    View in: PubMed
    Score: 0.096
  6. Immunohistochemistry of the Carotid Body. Methods Mol Biol. 2018; 1742:155-166.
    View in: PubMed
    Score: 0.096
  7. CaV3.2 T-type Ca2+ channels mediate the augmented calcium influx in carotid body glomus cells by chronic intermittent hypoxia. J Neurophysiol. 2016 Jan 01; 115(1):345-54.
    View in: PubMed
    Score: 0.083
  8. HIF-1a activation by intermittent hypoxia requires NADPH oxidase stimulation by xanthine oxidase. PLoS One. 2015; 10(3):e0119762.
    View in: PubMed
    Score: 0.079
  9. Carotid Body Chemoreflex Mediates Intermittent Hypoxia-Induced Oxidative Stress in the Adrenal Medulla. Adv Exp Med Biol. 2015; 860:195-9.
    View in: PubMed
    Score: 0.078
  10. CaV3.2 T-type Ca²? channels in H2S-mediated hypoxic response of the carotid body. Am J Physiol Cell Physiol. 2015 Jan 15; 308(2):C146-54.
    View in: PubMed
    Score: 0.077
  11. Regulation of hypoxia-inducible factor-a isoforms and redox state by carotid body neural activity in rats. J Physiol. 2014 Sep 01; 592(17):3841-58.
    View in: PubMed
    Score: 0.075
  12. Hypoxia-inducible factors regulate human and rat cystathionine ß-synthase gene expression. Biochem J. 2014 Mar 01; 458(2):203-11.
    View in: PubMed
    Score: 0.073
  13. Inherent variations in CO-H2S-mediated carotid body O2 sensing mediate hypertension and pulmonary edema. Proc Natl Acad Sci U S A. 2014 Jan 21; 111(3):1174-9.
    View in: PubMed
    Score: 0.073
  14. Xanthine oxidase mediates hypoxia-inducible factor-2a degradation by intermittent hypoxia. PLoS One. 2013; 8(10):e75838.
    View in: PubMed
    Score: 0.071
  15. Role of oxidative stress-induced endothelin-converting enzyme activity in the alteration of carotid body function by chronic intermittent hypoxia. Exp Physiol. 2013 Nov; 98(11):1620-30.
    View in: PubMed
    Score: 0.071
  16. Mutual antagonism between hypoxia-inducible factors 1a and 2a regulates oxygen sensing and cardio-respiratory homeostasis. Proc Natl Acad Sci U S A. 2013 May 07; 110(19):E1788-96.
    View in: PubMed
    Score: 0.069
  17. Endogenous H2S is required for hypoxic sensing by carotid body glomus cells. Am J Physiol Cell Physiol. 2012 Nov 01; 303(9):C916-23.
    View in: PubMed
    Score: 0.065
  18. Epigenetic regulation of hypoxic sensing disrupts cardiorespiratory homeostasis. Proc Natl Acad Sci U S A. 2012 Feb 14; 109(7):2515-20.
    View in: PubMed
    Score: 0.063
  19. Hypoxia-inducible factor 1 mediates increased expression of NADPH oxidase-2 in response to intermittent hypoxia. J Cell Physiol. 2011 Nov; 226(11):2925-33.
    View in: PubMed
    Score: 0.063
  20. Endothelin-1 mediates attenuated carotid baroreceptor activity by intermittent hypoxia. J Appl Physiol (1985). 2012 Jan; 112(1):187-96.
    View in: PubMed
    Score: 0.062
  21. Angiotensin II evokes sensory long-term facilitation of the carotid body via NADPH oxidase. J Appl Physiol (1985). 2011 Oct; 111(4):964-70.
    View in: PubMed
    Score: 0.061
  22. NADPH oxidase 2 mediates intermittent hypoxia-induced mitochondrial complex I inhibition: relevance to blood pressure changes in rats. Antioxid Redox Signal. 2011 Feb 15; 14(4):533-42.
    View in: PubMed
    Score: 0.058
  23. NADPH oxidase-dependent regulation of T-type Ca2+ channels and ryanodine receptors mediate the augmented exocytosis of catecholamines from intermittent hypoxia-treated neonatal rat chromaffin cells. J Neurosci. 2010 Aug 11; 30(32):10763-72.
    View in: PubMed
    Score: 0.057
  24. Neonatal intermittent hypoxia impairs neuronal nicotinic receptor expression and function in adrenal chromaffin cells. Am J Physiol Cell Physiol. 2010 Aug; 299(2):C381-8.
    View in: PubMed
    Score: 0.057
  25. H2S mediates O2 sensing in the carotid body. Proc Natl Acad Sci U S A. 2010 Jun 08; 107(23):10719-24.
    View in: PubMed
    Score: 0.057
  26. Refinement of telemetry for measuring blood pressure in conscious rats. J Am Assoc Lab Anim Sci. 2009 May; 48(3):268-71.
    View in: PubMed
    Score: 0.053
  27. NADPH oxidase is required for the sensory plasticity of the carotid body by chronic intermittent hypoxia. J Neurosci. 2009 Apr 15; 29(15):4903-10.
    View in: PubMed
    Score: 0.052
  28. Neonatal intermittent hypoxia leads to long-lasting facilitation of acute hypoxia-evoked catecholamine secretion from rat chromaffin cells. J Neurophysiol. 2009 Jun; 101(6):2837-46.
    View in: PubMed
    Score: 0.052
  29. Intermittent hypoxia degrades HIF-2alpha via calpains resulting in oxidative stress: implications for recurrent apnea-induced morbidities. Proc Natl Acad Sci U S A. 2009 Jan 27; 106(4):1199-204.
    View in: PubMed
    Score: 0.051
  30. Contrasting effects of intermittent and continuous hypoxia on low O(2) evoked catecholamine secretion from neonatal rat chromaffin cells. Adv Exp Med Biol. 2009; 648:345-9.
    View in: PubMed
    Score: 0.051
  31. Reactive oxygen species-dependent endothelin signaling is required for augmented hypoxic sensory response of the neonatal carotid body by intermittent hypoxia. Am J Physiol Regul Integr Comp Physiol. 2009 Mar; 296(3):R735-42.
    View in: PubMed
    Score: 0.051
  32. Induction of HIF-1alpha expression by intermittent hypoxia: involvement of NADPH oxidase, Ca2+ signaling, prolyl hydroxylases, and mTOR. J Cell Physiol. 2008 Dec; 217(3):674-85.
    View in: PubMed
    Score: 0.051
  33. Comparative analysis of neonatal and adult rat carotid body responses to chronic intermittent hypoxia. J Appl Physiol (1985). 2008 May; 104(5):1287-94.
    View in: PubMed
    Score: 0.048
  34. Acute intermittent hypoxia increases both phrenic and sympathetic nerve activities in the rat. Exp Physiol. 2007 Jan; 92(1):87-97.
    View in: PubMed
    Score: 0.044
  35. 5-HT evokes sensory long-term facilitation of rodent carotid body via activation of NADPH oxidase. J Physiol. 2006 Oct 01; 576(Pt 1):289-95.
    View in: PubMed
    Score: 0.043
  36. Acute lung injury augments hypoxic ventilatory response in the absence of systemic hypoxemia. J Appl Physiol (1985). 2006 Dec; 101(6):1795-802.
    View in: PubMed
    Score: 0.043
  37. Chronic intermittent hypoxia induces hypoxia-evoked catecholamine efflux in adult rat adrenal medulla via oxidative stress. J Physiol. 2006 Aug 15; 575(Pt 1):229-39.
    View in: PubMed
    Score: 0.043
  38. Modulation of the hypoxic sensory response of the carotid body by 5-hydroxytryptamine: role of the 5-HT2 receptor. Respir Physiol Neurobiol. 2005 Feb 15; 145(2-3):135-42.
    View in: PubMed
    Score: 0.039
  39. Ca2+/calmodulin kinase-dependent activation of hypoxia inducible factor 1 transcriptional activity in cells subjected to intermittent hypoxia. J Biol Chem. 2005 Feb 11; 280(6):4321-8.
    View in: PubMed
    Score: 0.039
  40. Intermittent hypoxia augments carotid body and ventilatory response to hypoxia in neonatal rat pups. J Appl Physiol (1985). 2004 Nov; 97(5):2020-5.
    View in: PubMed
    Score: 0.038
  41. Role of oxidative stress in intermittent hypoxia-induced immediate early gene activation in rat PC12 cells. J Physiol. 2004 Jun 15; 557(Pt 3):773-83.
    View in: PubMed
    Score: 0.037
  42. Entrainment pattern between sympathetic and phrenic nerve activities in the Sprague-Dawley rat: hypoxia-evoked sympathetic activity during expiration. Am J Physiol Regul Integr Comp Physiol. 2004 Jun; 286(6):R1121-8.
    View in: PubMed
    Score: 0.037
  43. Transcriptomic Analysis of Postnatal Rat Carotid Body Development. Genes (Basel). 2024 02 27; 15(3).
    View in: PubMed
    Score: 0.037
  44. P300/CBP Regulates HIF-1-Dependent Sympathetic Activation and Hypertension by Intermittent Hypoxia. Am J Respir Cell Mol Biol. 2024 Feb; 70(2):110-118.
    View in: PubMed
    Score: 0.037
  45. Detection of oxygen sensing during intermittent hypoxia. Methods Enzymol. 2004; 381:107-20.
    View in: PubMed
    Score: 0.036
  46. Effect of two paradigms of chronic intermittent hypoxia on carotid body sensory activity. J Appl Physiol (1985). 2004 Mar; 96(3):1236-42; discussion 1196.
    View in: PubMed
    Score: 0.036
  47. Induction of sensory long-term facilitation in the carotid body by intermittent hypoxia: implications for recurrent apneas. Proc Natl Acad Sci U S A. 2003 Aug 19; 100(17):10073-8.
    View in: PubMed
    Score: 0.035
  48. Activation of tyrosine hydroxylase by intermittent hypoxia: involvement of serine phosphorylation. J Appl Physiol (1985). 2003 Aug; 95(2):536-44.
    View in: PubMed
    Score: 0.035
  49. Reactive oxygen species in the plasticity of respiratory behavior elicited by chronic intermittent hypoxia. J Appl Physiol (1985). 2003 Jun; 94(6):2342-9.
    View in: PubMed
    Score: 0.034
  50. Systemic and cellular responses to intermittent hypoxia: evidence for oxidative stress and mitochondrial dysfunction. Adv Exp Med Biol. 2003; 536:559-64.
    View in: PubMed
    Score: 0.034
  51. Protein phosphatase 1 regulates reactive oxygen species-dependent degradation of histone deacetylase 5 by intermittent hypoxia. Am J Physiol Cell Physiol. 2022 08 01; 323(2):C423-C431.
    View in: PubMed
    Score: 0.033
  52. Ventilatory changes during intermittent hypoxia: importance of pattern and duration. High Alt Med Biol. 2002; 3(2):195-204.
    View in: PubMed
    Score: 0.032
  53. Selected Contribution: Improved anoxic tolerance in rat diaphragm following intermittent hypoxia. J Appl Physiol (1985). 2001 Jun; 90(6):2508-13.
    View in: PubMed
    Score: 0.030
  54. Gene regulation during intermittent hypoxia: evidence for the involvement of reactive oxygen species. Adv Exp Med Biol. 2001; 499:297-302.
    View in: PubMed
    Score: 0.030
  55. Chronic intermittent hypoxia enhances carotid body chemoreceptor response to low oxygen. Adv Exp Med Biol. 2001; 499:33-8.
    View in: PubMed
    Score: 0.030
  56. L-type Ca(2+) channel activation regulates induction of c-fos transcription by hypoxia. J Appl Physiol (1985). 2000 May; 88(5):1898-906.
    View in: PubMed
    Score: 0.028
  57. Intracellular pathways linking hypoxia to activation of c-fos and AP-1. Adv Exp Med Biol. 2000; 475:101-9.
    View in: PubMed
    Score: 0.028
  58. Dual influence of nitric oxide on gene regulation during hypoxia. Adv Exp Med Biol. 2000; 475:285-92.
    View in: PubMed
    Score: 0.028
  59. Role of c-fos in hypoxia-induced AP-1 cis-element activity and tyrosine hydroxylase gene expression. Brain Res Mol Brain Res. 1998 Aug 15; 59(1):74-83.
    View in: PubMed
    Score: 0.025
  60. Release of dopamine and norepinephrine by hypoxia from PC-12 cells. Am J Physiol. 1998 06; 274(6):C1592-600.
    View in: PubMed
    Score: 0.025
  61. Ca2+ current in rabbit carotid body glomus cells is conducted by multiple types of high-voltage-activated Ca2+ channels. J Neurophysiol. 1997 Nov; 78(5):2467-74.
    View in: PubMed
    Score: 0.024
  62. Activation of nitric oxide synthase gene expression by hypoxia in central and peripheral neurons. Brain Res Mol Brain Res. 1996 Dec 31; 43(1-2):341-6.
    View in: PubMed
    Score: 0.022
  63. Heterogeneity in cytosolic calcium responses to hypoxia in carotid body cells. Brain Res. 1996 Jan 15; 706(2):297-302.
    View in: PubMed
    Score: 0.021
  64. Induction of immediate early response genes by hypoxia. Possible molecular bases for systems adaptation to low pO2. Adv Exp Med Biol. 1996; 410:127-34.
    View in: PubMed
    Score: 0.021
  65. Regulation of neuronal nitric oxide synthase gene expression by hypoxia. Role of nitric oxide in respiratory adaptation to low pO2. Adv Exp Med Biol. 1996; 410:345-8.
    View in: PubMed
    Score: 0.021
  66. Cell selective induction and transcriptional activation of immediate early genes by hypoxia. Brain Res. 1995 Oct 30; 697(1-2):266-70.
    View in: PubMed
    Score: 0.021
  67. Carbon monoxide: a role in carotid body chemoreception. Proc Natl Acad Sci U S A. 1995 Mar 14; 92(6):1994-7.
    View in: PubMed
    Score: 0.020
  68. Tachykinin antagonists in carotid body responses to hypoxia and substance P in the rat. Respir Physiol. 1994 Mar; 95(3):295-310.
    View in: PubMed
    Score: 0.018
  69. Role of substance P in rat carotid body responses to hypoxia and capsaicin. Adv Exp Med Biol. 1993; 337:265-70.
    View in: PubMed
    Score: 0.017
  70. NADPH oxidase-derived H(2)O(2) contributes to angiotensin II-induced aldosterone synthesis in human and rat adrenal cortical cells. Antioxid Redox Signal. 2012 Aug 01; 17(3):445-59.
    View in: PubMed
    Score: 0.016
  71. Enhanced neuropeptide Y synthesis during intermittent hypoxia in the rat adrenal medulla: role of reactive oxygen species-dependent alterations in precursor peptide processing. Antioxid Redox Signal. 2011 Apr 01; 14(7):1179-90.
    View in: PubMed
    Score: 0.015
  72. Post-translational modification of glutamic acid decarboxylase 67 by intermittent hypoxia: evidence for the involvement of dopamine D1 receptor signaling. J Neurochem. 2010 Dec; 115(6):1568-78.
    View in: PubMed
    Score: 0.015
  73. Substance P and mitochondrial oxygen consumption: evidence for a direct intracellular role for the peptide. Peptides. 1989 Sep-Oct; 10(5):1003-6.
    View in: PubMed
    Score: 0.013
  74. Pattern-specific sustained activation of tyrosine hydroxylase by intermittent hypoxia: role of reactive oxygen species-dependent downregulation of protein phosphatase 2A and upregulation of protein kinases. Antioxid Redox Signal. 2009 Aug; 11(8):1777-89.
    View in: PubMed
    Score: 0.013
  75. Intermittent hypoxia activates peptidylglycine alpha-amidating monooxygenase in rat brain stem via reactive oxygen species-mediated proteolytic processing. J Appl Physiol (1985). 2009 Jan; 106(1):12-9.
    View in: PubMed
    Score: 0.013
  76. Influence of adrenaline and hypoxia on rat muscle receptors in vitro. Prog Brain Res. 1988; 74:91-7.
    View in: PubMed
    Score: 0.012
  77. Secretion of brain-derived neurotrophic factor from PC12 cells in response to oxidative stress requires autocrine dopamine signaling. J Neurochem. 2006 Feb; 96(3):694-705.
    View in: PubMed
    Score: 0.010
  78. Facilitation of dopamine and acetylcholine release by intermittent hypoxia in PC12 cells: involvement of calcium and reactive oxygen species. J Appl Physiol (1985). 2004 Mar; 96(3):1206-15; discussion 1196.
    View in: PubMed
    Score: 0.009
  79. Nitric oxide and ventilatory response to hypoxia. Respir Physiol. 1995 Sep; 101(3):257-66.
    View in: PubMed
    Score: 0.005
  80. Possible genomic mechanism involved in control systems responses to hypoxia. Adv Exp Med Biol. 1995; 393:89-94.
    View in: PubMed
    Score: 0.005
  81. Low PO2 dependency of neutral endopeptidase and acetylcholinesterase activities of the rat carotid body. Adv Exp Med Biol. 1994; 360:217-20.
    View in: PubMed
    Score: 0.005
  82. Integration of cardiorespiratory responses in the ventrolateral medulla. Prog Brain Res. 1989; 81:215-20.
    View in: PubMed
    Score: 0.003
  83. Respiratory and vasomotor responses to focal cooling of the ventral medullary surface (VMS) of the rat. Respir Physiol. 1988 Oct; 74(1):35-47.
    View in: PubMed
    Score: 0.003
Connection Strength

The connection strength for concepts is the sum of the scores for each matching publication.

Publication scores are based on many factors, including how long ago they were written and whether the person is a first or senior author.