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Connection

Nanduri R. Prabhakar to Cell Hypoxia

This is a "connection" page, showing publications Nanduri R. Prabhakar has written about Cell Hypoxia.
Connection Strength

3.796
  1. Lysine demethylase KDM6B regulates HIF-1a-mediated systemic and cellular responses to intermittent hypoxia. Physiol Genomics. 2021 09 01; 53(9):385-394.
    View in: PubMed
    Score: 0.761
  2. Xanthine oxidase mediates hypoxia-inducible factor-2a degradation by intermittent hypoxia. PLoS One. 2013; 8(10):e75838.
    View in: PubMed
    Score: 0.443
  3. O2 sensing at the mammalian carotid body: why multiple O2 sensors and multiple transmitters? Exp Physiol. 2006 Jan; 91(1):17-23.
    View in: PubMed
    Score: 0.255
  4. Oxidative stress in the systemic and cellular responses to intermittent hypoxia. Biol Chem. 2004 Mar-Apr; 385(3-4):217-21.
    View in: PubMed
    Score: 0.228
  5. Augmentation of L-type calcium current by hypoxia in rabbit carotid body glomus cells: evidence for a PKC-sensitive pathway. J Neurophysiol. 2000 Sep; 84(3):1636-44.
    View in: PubMed
    Score: 0.179
  6. Dual influence of nitric oxide on gene regulation during hypoxia. Adv Exp Med Biol. 2000; 475:285-92.
    View in: PubMed
    Score: 0.171
  7. Augmentation of calcium current by hypoxia in carotid body glomus cells. Adv Exp Med Biol. 2000; 475:589-99.
    View in: PubMed
    Score: 0.171
  8. Release of dopamine and norepinephrine by hypoxia from PC-12 cells. Am J Physiol. 1998 06; 274(6):C1592-600.
    View in: PubMed
    Score: 0.153
  9. Measurement of Sensory Nerve Activity from the Carotid Body. Methods Mol Biol. 2018; 1742:115-124.
    View in: PubMed
    Score: 0.149
  10. Heterogeneity in cytosolic calcium responses to hypoxia in carotid body cells. Brain Res. 1996 Jan 15; 706(2):297-302.
    View in: PubMed
    Score: 0.130
  11. CaV3.2 T-type Ca2+ channels mediate the augmented calcium influx in carotid body glomus cells by chronic intermittent hypoxia. J Neurophysiol. 2016 Jan 01; 115(1):345-54.
    View in: PubMed
    Score: 0.128
  12. HIF-1a activation by intermittent hypoxia requires NADPH oxidase stimulation by xanthine oxidase. PLoS One. 2015; 10(3):e0119762.
    View in: PubMed
    Score: 0.122
  13. Peripheral chemoreceptors: function and plasticity of the carotid body. Compr Physiol. 2012 Jan; 2(1):141-219.
    View in: PubMed
    Score: 0.098
  14. Hypoxia inhibits maturation and trafficking of hERG K(+) channel protein: Role of Hsp90 and ROS. Biochem Biophys Res Commun. 2009 Oct 16; 388(2):212-6.
    View in: PubMed
    Score: 0.083
  15. Neonatal intermittent hypoxia leads to long-lasting facilitation of acute hypoxia-evoked catecholamine secretion from rat chromaffin cells. J Neurophysiol. 2009 Jun; 101(6):2837-46.
    View in: PubMed
    Score: 0.081
  16. Intermittent hypoxia degrades HIF-2alpha via calpains resulting in oxidative stress: implications for recurrent apnea-induced morbidities. Proc Natl Acad Sci U S A. 2009 Jan 27; 106(4):1199-204.
    View in: PubMed
    Score: 0.080
  17. Induction of HIF-1alpha expression by intermittent hypoxia: involvement of NADPH oxidase, Ca2+ signaling, prolyl hydroxylases, and mTOR. J Cell Physiol. 2008 Dec; 217(3):674-85.
    View in: PubMed
    Score: 0.079
  18. Protein phosphatase 1 regulates reactive oxygen species-dependent degradation of histone deacetylase 5 by intermittent hypoxia. Am J Physiol Cell Physiol. 2022 08 01; 323(2):C423-C431.
    View in: PubMed
    Score: 0.051
  19. Gene regulation during intermittent hypoxia: evidence for the involvement of reactive oxygen species. Adv Exp Med Biol. 2001; 499:297-302.
    View in: PubMed
    Score: 0.046
  20. Intracellular pathways linking hypoxia to activation of c-fos and AP-1. Adv Exp Med Biol. 2000; 475:101-9.
    View in: PubMed
    Score: 0.043
  21. Chemosensing at the carotid body. Involvement of a HERG-like potassium current in glomus cells. Adv Exp Med Biol. 2000; 475:241-8.
    View in: PubMed
    Score: 0.043
  22. Hypoxia induced hERG trafficking defect linked to cell cycle arrest in SH-SY5Y cells. PLoS One. 2019; 14(4):e0215905.
    View in: PubMed
    Score: 0.041
  23. NO and CO as second messengers in oxygen sensing in the carotid body. Respir Physiol. 1999 Apr 01; 115(2):161-8.
    View in: PubMed
    Score: 0.041
  24. Role of c-fos in hypoxia-induced AP-1 cis-element activity and tyrosine hydroxylase gene expression. Brain Res Mol Brain Res. 1998 Aug 15; 59(1):74-83.
    View in: PubMed
    Score: 0.039
  25. Induction of immediate early response genes by hypoxia. Possible molecular bases for systems adaptation to low pO2. Adv Exp Med Biol. 1996; 410:127-34.
    View in: PubMed
    Score: 0.032
  26. Calpain activation by ROS mediates human ether-a-go-go-related gene protein degradation by intermittent hypoxia. Am J Physiol Cell Physiol. 2016 Mar 01; 310(5):C329-36.
    View in: PubMed
    Score: 0.032
  27. Cell selective induction and transcriptional activation of immediate early genes by hypoxia. Brain Res. 1995 Oct 30; 697(1-2):266-70.
    View in: PubMed
    Score: 0.032
  28. TET1-mediated hydroxymethylation facilitates hypoxic gene induction in neuroblastoma. Cell Rep. 2014 Jun 12; 7(5):1343-1352.
    View in: PubMed
    Score: 0.029
  29. Enhanced neuropeptide Y synthesis during intermittent hypoxia in the rat adrenal medulla: role of reactive oxygen species-dependent alterations in precursor peptide processing. Antioxid Redox Signal. 2011 Apr 01; 14(7):1179-90.
    View in: PubMed
    Score: 0.023
  30. Post-translational modification of glutamic acid decarboxylase 67 by intermittent hypoxia: evidence for the involvement of dopamine D1 receptor signaling. J Neurochem. 2010 Dec; 115(6):1568-78.
    View in: PubMed
    Score: 0.023
  31. Facilitation of dopamine and acetylcholine release by intermittent hypoxia in PC12 cells: involvement of calcium and reactive oxygen species. J Appl Physiol (1985). 2004 Mar; 96(3):1206-15; discussion 1196.
    View in: PubMed
    Score: 0.014
Connection Strength

The connection strength for concepts is the sum of the scores for each matching publication.

Publication scores are based on many factors, including how long ago they were written and whether the person is a first or senior author.