Michael Thirman to Oncogene Proteins, Fusion
This is a "connection" page, showing publications Michael Thirman has written about Oncogene Proteins, Fusion.
Connection Strength
1.331
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MLL fusion proteins preferentially regulate a subset of wild-type MLL target genes in the leukemic genome. Blood. 2011 Jun 23; 117(25):6895-905.
Score: 0.321
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Consistent deregulation of gene expression between human and murine MLL rearrangement leukemias. Cancer Res. 2009 Feb 01; 69(3):1109-16.
Score: 0.275
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EAF1, a novel ELL-associated factor that is delocalized by expression of the MLL-ELL fusion protein. Blood. 2001 Jul 01; 98(1):201-9.
Score: 0.163
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Retrovirus-mediated gene transfer of MLL-ELL transforms primary myeloid progenitors and causes acute myeloid leukemias in mice. Proc Natl Acad Sci U S A. 2000 Sep 26; 97(20):10984-9.
Score: 0.154
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The full transforming capacity of MLL-Af4 is interlinked with lymphoid lineage commitment. Blood. 2017 08 17; 130(7):903-907.
Score: 0.123
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Instructive Role of MLL-Fusion Proteins Revealed by a Model of t(4;11) Pro-B Acute Lymphoblastic Leukemia. Cancer Cell. 2016 Nov 14; 30(5):737-749.
Score: 0.118
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ELL and EAF1 are Cajal body components that are disrupted in MLL-ELL leukemia. Mol Biol Cell. 2003 Apr; 14(4):1517-28.
Score: 0.046
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ELL-associated factor 2 (EAF2), a functional homolog of EAF1 with alternative ELL binding properties. Blood. 2003 Mar 15; 101(6):2355-62.
Score: 0.045
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Epigenetic regulator genes direct lineage switching in MLL/AF4 leukemia. Blood. 2022 10 27; 140(17):1875-1890.
Score: 0.045
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The elongation domain of ELL is dispensable but its ELL-associated factor 1 interaction domain is essential for MLL-ELL-induced leukemogenesis. Mol Cell Biol. 2001 Aug; 21(16):5678-87.
Score: 0.041