Bana Jabri to T-Lymphocytes, Cytotoxic
This is a "connection" page, showing publications Bana Jabri has written about T-Lymphocytes, Cytotoxic.
Connection Strength
2.491
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Cysteinyl leukotrienes mediate lymphokine killer activity induced by NKG2D and IL-15 in cytotoxic T cells during celiac disease. J Exp Med. 2015 Sep 21; 212(10):1487-95.
Score: 0.481
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Cytosolic PLA2 is required for CTL-mediated immunopathology of celiac disease via NKG2D and IL-15. J Exp Med. 2009 Mar 16; 206(3):707-19.
Score: 0.307
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Human CD8+ intraepithelial lymphocytes: a unique model to study the regulation of effector cytotoxic T lymphocytes in tissue. Immunol Rev. 2007 Feb; 215:202-14.
Score: 0.266
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Reprogramming of CTLs into natural killer-like cells in celiac disease. J Exp Med. 2006 May 15; 203(5):1343-55.
Score: 0.253
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NKG2 receptor-mediated regulation of effector CTL functions in the human tissue microenvironment. Curr Top Microbiol Immunol. 2006; 298:139-56.
Score: 0.247
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Coordinated induction by IL15 of a TCR-independent NKG2D signaling pathway converts CTL into lymphokine-activated killer cells in celiac disease. Immunity. 2004 Sep; 21(3):357-66.
Score: 0.225
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TCR specificity dictates CD94/NKG2A expression by human CTL. Immunity. 2002 Oct; 17(4):487-99.
Score: 0.197
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NKG2D receptors induced by IL-15 costimulate CD28-negative effector CTL in the tissue microenvironment. J Immunol. 2001 Nov 15; 167(10):5527-30.
Score: 0.185
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Tissue alarmins and adaptive cytokine induce dynamic and distinct transcriptional responses in tissue-resident intraepithelial cytotoxic T lymphocytes. J Autoimmun. 2020 03; 108:102422.
Score: 0.164
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Distinct and Synergistic Contributions of Epithelial Stress and Adaptive Immunity to Functions of Intraepithelial Killer Cells and Active Celiac Disease. Gastroenterology. 2015 Sep; 149(3):681-91.e10.
Score: 0.118
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Tet2 deficiency drives liver microbiome dysbiosis triggering Tc1 cell autoimmune hepatitis. Cell Host Microbe. 2022 07 13; 30(7):1003-1019.e10.
Score: 0.048