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Connection

Jayasri Nanduri to Hypoxia

This is a "connection" page, showing publications Jayasri Nanduri has written about Hypoxia.
Connection Strength

7.783
  1. Protein phosphatase 1 regulates reactive oxygen species-dependent degradation of histone deacetylase 5 by intermittent hypoxia. Am J Physiol Cell Physiol. 2022 08 01; 323(2):C423-C431.
    View in: PubMed
    Score: 0.510
  2. Adaptive cardiorespiratory changes to chronic continuous and intermittent hypoxia. Handb Clin Neurol. 2022; 188:103-123.
    View in: PubMed
    Score: 0.494
  3. Lysine demethylase KDM6B regulates HIF-1a-mediated systemic and cellular responses to intermittent hypoxia. Physiol Genomics. 2021 09 01; 53(9):385-394.
    View in: PubMed
    Score: 0.479
  4. Hypoxia-inducible factor-1 mediates pancreatic ß-cell dysfunction by intermittent hypoxia. Am J Physiol Cell Physiol. 2020 11 01; 319(5):C922-C932.
    View in: PubMed
    Score: 0.452
  5. Recent advances in understanding the physiology of hypoxic sensing by the carotid body. F1000Res. 2018; 7.
    View in: PubMed
    Score: 0.399
  6. Reactive oxygen radicals and gaseous transmitters in carotid body activation by intermittent hypoxia. Cell Tissue Res. 2018 05; 372(2):427-431.
    View in: PubMed
    Score: 0.378
  7. Epigenetic changes by DNA methylation in chronic and intermittent hypoxia. Am J Physiol Lung Cell Mol Physiol. 2017 Dec 01; 313(6):L1096-L1100.
    View in: PubMed
    Score: 0.365
  8. Epigenetic regulation of redox state mediates persistent cardiorespiratory abnormalities after long-term intermittent hypoxia. J Physiol. 2017 01 01; 595(1):63-77.
    View in: PubMed
    Score: 0.343
  9. Peripheral chemoreception and arterial pressure responses to intermittent hypoxia. Compr Physiol. 2015 Apr; 5(2):561-77.
    View in: PubMed
    Score: 0.309
  10. Impairment of pancreatic ß-cell function by chronic intermittent hypoxia. Exp Physiol. 2013 Sep; 98(9):1376-85.
    View in: PubMed
    Score: 0.272
  11. Epigenetic regulation of hypoxic sensing disrupts cardiorespiratory homeostasis. Proc Natl Acad Sci U S A. 2012 Feb 14; 109(7):2515-20.
    View in: PubMed
    Score: 0.247
  12. Intermittent hypoxia augments acute hypoxic sensing via HIF-mediated ROS. Respir Physiol Neurobiol. 2010 Dec 31; 174(3):230-4.
    View in: PubMed
    Score: 0.226
  13. Intermittent hypoxia-mediated plasticity of acute O2 sensing requires altered red-ox regulation by HIF-1 and HIF-2. Ann N Y Acad Sci. 2009 Oct; 1177:162-8.
    View in: PubMed
    Score: 0.211
  14. Transcriptional responses to intermittent hypoxia. Respir Physiol Neurobiol. 2008 Dec 10; 164(1-2):277-81.
    View in: PubMed
    Score: 0.200
  15. Cellular mechanisms associated with intermittent hypoxia. Essays Biochem. 2007; 43:91-104.
    View in: PubMed
    Score: 0.175
  16. Signal Transduction Pathway Mediating Carotid Body Dependent Sympathetic Activation and Hypertension by Chronic Intermittent Hypoxia. Function (Oxf). 2025 Feb 12; 6(1).
    View in: PubMed
    Score: 0.153
  17. Adrenal epinephrine facilitates erythropoietin gene activation by hypoxia through ß2 adrenergic receptor interaction with Hif-2a. Am J Physiol Regul Integr Comp Physiol. 2025 Jan 01; 328(1):R75-R80.
    View in: PubMed
    Score: 0.151
  18. Transcriptomic Analysis of Postnatal Rat Carotid Body Development. Genes (Basel). 2024 02 27; 15(3).
    View in: PubMed
    Score: 0.143
  19. P300/CBP Regulates HIF-1-Dependent Sympathetic Activation and Hypertension by Intermittent Hypoxia. Am J Respir Cell Mol Biol. 2024 Feb; 70(2):110-118.
    View in: PubMed
    Score: 0.143
  20. Carotid body hypersensitivity in intermittent hypoxia and obtructive sleep apnoea. J Physiol. 2023 12; 601(24):5481-5494.
    View in: PubMed
    Score: 0.136
  21. Role of olfactory receptor78 in carotid body-dependent sympathetic activation and hypertension in murine models of chronic intermittent hypoxia. J Neurophysiol. 2021 06 01; 125(6):2054-2067.
    View in: PubMed
    Score: 0.118
  22. Gaseous transmitter regulation of hypoxia-evoked catecholamine secretion from murine adrenal chromaffin cells. J Neurophysiol. 2021 05 01; 125(5):1533-1542.
    View in: PubMed
    Score: 0.117
  23. Hypoxia-inducible factors and obstructive sleep apnea. J Clin Invest. 2020 10 01; 130(10):5042-5051.
    View in: PubMed
    Score: 0.113
  24. Olfactory receptor 78 participates in carotid body response to a wide range of low O2 levels but not severe hypoxia. J Neurophysiol. 2020 05 01; 123(5):1886-1895.
    View in: PubMed
    Score: 0.109
  25. Long-term facilitation of catecholamine secretion from adrenal chromaffin cells of neonatal rats by chronic intermittent hypoxia. J Neurophysiol. 2019 11 01; 122(5):1874-1883.
    View in: PubMed
    Score: 0.105
  26. DNA methylation in the central and efferent limbs of the chemoreflex requires carotid body neural activity. J Physiol. 2018 08; 596(15):3087-3100.
    View in: PubMed
    Score: 0.094
  27. CaV3.2 T-type Ca2+ channels mediate the augmented calcium influx in carotid body glomus cells by chronic intermittent hypoxia. J Neurophysiol. 2016 Jan 01; 115(1):345-54.
    View in: PubMed
    Score: 0.081
  28. Hypoxia-inducible factors and hypertension: lessons from sleep apnea syndrome. J Mol Med (Berl). 2015 May; 93(5):473-80.
    View in: PubMed
    Score: 0.077
  29. Neuromolecular mechanisms mediating the effects of chronic intermittent hypoxia on adrenal medulla. Respir Physiol Neurobiol. 2015 Apr; 209:115-9.
    View in: PubMed
    Score: 0.076
  30. Epigenetic Regulation of Carotid Body Oxygen Sensing: Clinical Implications. Adv Exp Med Biol. 2015; 860:1-8.
    View in: PubMed
    Score: 0.076
  31. Carotid Body Chemoreflex Mediates Intermittent Hypoxia-Induced Oxidative Stress in the Adrenal Medulla. Adv Exp Med Biol. 2015; 860:195-9.
    View in: PubMed
    Score: 0.076
  32. CaV3.2 T-type Ca²? channels in H2S-mediated hypoxic response of the carotid body. Am J Physiol Cell Physiol. 2015 Jan 15; 308(2):C146-54.
    View in: PubMed
    Score: 0.075
  33. Regulation of hypoxia-inducible factor-a isoforms and redox state by carotid body neural activity in rats. J Physiol. 2014 Sep 01; 592(17):3841-58.
    View in: PubMed
    Score: 0.073
  34. Role of oxidative stress-induced endothelin-converting enzyme activity in the alteration of carotid body function by chronic intermittent hypoxia. Exp Physiol. 2013 Nov; 98(11):1620-30.
    View in: PubMed
    Score: 0.069
  35. Endogenous H2S is required for hypoxic sensing by carotid body glomus cells. Am J Physiol Cell Physiol. 2012 Nov 01; 303(9):C916-23.
    View in: PubMed
    Score: 0.064
  36. Hypoxia-inducible factor 1 mediates increased expression of NADPH oxidase-2 in response to intermittent hypoxia. J Cell Physiol. 2011 Nov; 226(11):2925-33.
    View in: PubMed
    Score: 0.061
  37. Endothelin-1 mediates attenuated carotid baroreceptor activity by intermittent hypoxia. J Appl Physiol (1985). 2012 Jan; 112(1):187-96.
    View in: PubMed
    Score: 0.061
  38. Hypoxia-inducible factor 2a (HIF-2a) heterozygous-null mice exhibit exaggerated carotid body sensitivity to hypoxia, breathing instability, and hypertension. Proc Natl Acad Sci U S A. 2011 Feb 15; 108(7):3065-70.
    View in: PubMed
    Score: 0.058
  39. NADPH oxidase 2 mediates intermittent hypoxia-induced mitochondrial complex I inhibition: relevance to blood pressure changes in rats. Antioxid Redox Signal. 2011 Feb 15; 14(4):533-42.
    View in: PubMed
    Score: 0.057
  40. NADPH oxidase-dependent regulation of T-type Ca2+ channels and ryanodine receptors mediate the augmented exocytosis of catecholamines from intermittent hypoxia-treated neonatal rat chromaffin cells. J Neurosci. 2010 Aug 11; 30(32):10763-72.
    View in: PubMed
    Score: 0.056
  41. Long-term regulation of carotid body function: acclimatization and adaptation--invited article. Adv Exp Med Biol. 2009; 648:307-17.
    View in: PubMed
    Score: 0.050
  42. Reactive oxygen species-dependent endothelin signaling is required for augmented hypoxic sensory response of the neonatal carotid body by intermittent hypoxia. Am J Physiol Regul Integr Comp Physiol. 2009 Mar; 296(3):R735-42.
    View in: PubMed
    Score: 0.050
  43. ROS signaling in systemic and cellular responses to chronic intermittent hypoxia. Antioxid Redox Signal. 2007 Sep; 9(9):1397-403.
    View in: PubMed
    Score: 0.046
  44. Systemic, cellular and molecular analysis of chemoreflex-mediated sympathoexcitation by chronic intermittent hypoxia. Exp Physiol. 2007 Jan; 92(1):39-44.
    View in: PubMed
    Score: 0.043
  45. Ca2+/calmodulin kinase-dependent activation of hypoxia inducible factor 1 transcriptional activity in cells subjected to intermittent hypoxia. J Biol Chem. 2005 Feb 11; 280(6):4321-8.
    View in: PubMed
    Score: 0.038
  46. Hypoxia sensing requires H2S-dependent persulfidation of olfactory receptor 78. Sci Adv. 2023 07 07; 9(27):eadf3026.
    View in: PubMed
    Score: 0.034
  47. Carotid body responses to O2 and CO2 in hypoxia-tolerant naked mole rats. Acta Physiol (Oxf). 2022 10; 236(2):e13851.
    View in: PubMed
    Score: 0.032
  48. Protein kinase G-regulated production of H2S governs oxygen sensing. Sci Signal. 2015 Apr 21; 8(373):ra37.
    View in: PubMed
    Score: 0.019
  49. Intermittent hypoxia-induced endothelial barrier dysfunction requires ROS-dependent MAP kinase activation. Am J Physiol Cell Physiol. 2014 Apr 15; 306(8):C745-52.
    View in: PubMed
    Score: 0.018
  50. Inherent variations in CO-H2S-mediated carotid body O2 sensing mediate hypertension and pulmonary edema. Proc Natl Acad Sci U S A. 2014 Jan 21; 111(3):1174-9.
    View in: PubMed
    Score: 0.018
  51. Mutual antagonism between hypoxia-inducible factors 1a and 2a regulates oxygen sensing and cardio-respiratory homeostasis. Proc Natl Acad Sci U S A. 2013 May 07; 110(19):E1788-96.
    View in: PubMed
    Score: 0.017
  52. H2S mediates O2 sensing in the carotid body. Proc Natl Acad Sci U S A. 2010 Jun 08; 107(23):10719-24.
    View in: PubMed
    Score: 0.014
Connection Strength

The connection strength for concepts is the sum of the scores for each matching publication.

Publication scores are based on many factors, including how long ago they were written and whether the person is a first or senior author.