The University of Chicago Header Logo

Connection

Anthony Chang to Mice, Inbred C57BL

This is a "connection" page, showing publications Anthony Chang has written about Mice, Inbred C57BL.
Connection Strength

0.319
  1. Androgens contribute to sex bias of autoimmunity in mice by T cell-intrinsic regulation of Ptpn22 phosphatase expression. Nat Commun. 2024 Sep 03; 15(1):7688.
    View in: PubMed
    Score: 0.036
  2. Macrophage Depletion Reduces Disease Pathology in Factor H-Dependent Immune Complex-Mediated Glomerulonephritis. J Immunol Res. 2022; 2022:1737419.
    View in: PubMed
    Score: 0.030
  3. Bim suppresses the development of SLE by limiting myeloid inflammatory responses. J Exp Med. 2017 Dec 04; 214(12):3753-3773.
    View in: PubMed
    Score: 0.023
  4. Abrogation of immune complex glomerulonephritis by native carboxypeptidase and pharmacological antagonism of the C5a receptor. Cell Mol Immunol. 2016 09; 13(5):651-7.
    View in: PubMed
    Score: 0.019
  5. CD11b is protective in complement-mediated immune complex glomerulonephritis. Kidney Int. 2015 May; 87(5):930-9.
    View in: PubMed
    Score: 0.019
  6. Longitudinal changes in MRI markers in a reversible unilateral ureteral obstruction mouse model: preliminary experience. J Magn Reson Imaging. 2014 Apr; 39(4):835-41.
    View in: PubMed
    Score: 0.017
  7. TNF-mediated damage to glomerular endothelium is an important determinant of acute kidney injury in sepsis. Kidney Int. 2014 Jan; 85(1):72-81.
    View in: PubMed
    Score: 0.017
  8. Curcumin alleviates immune-complex-mediated glomerulonephritis in factor-H-deficient mice. Immunology. 2013 Jul; 139(3):328-37.
    View in: PubMed
    Score: 0.017
  9. The C5a receptor has a key role in immune complex glomerulonephritis in complement factor H-deficient mice. Kidney Int. 2012 Nov; 82(9):961-8.
    View in: PubMed
    Score: 0.016
  10. Abnormal immune complex processing and spontaneous glomerulonephritis in complement factor H-deficient mice with human complement receptor 1 on erythrocytes. J Immunol. 2010 Sep 15; 185(6):3759-67.
    View in: PubMed
    Score: 0.014
  11. Vitamin D receptor attenuates renal fibrosis by suppressing the renin-angiotensin system. J Am Soc Nephrol. 2010 Jun; 21(6):966-73.
    View in: PubMed
    Score: 0.013
  12. Transgenic overexpression of GLUT1 in mouse glomeruli produces renal disease resembling diabetic glomerulosclerosis. Am J Physiol Renal Physiol. 2010 Jul; 299(1):F99-F111.
    View in: PubMed
    Score: 0.013
  13. Combined vitamin D analog and AT1 receptor antagonist synergistically block the development of kidney disease in a model of type 2 diabetes. Kidney Int. 2010 Jun; 77(11):1000-9.
    View in: PubMed
    Score: 0.013
  14. Chronic kidney disease induced in mice by reversible unilateral ureteral obstruction is dependent on genetic background. Am J Physiol Renal Physiol. 2010 Apr; 298(4):F1024-32.
    View in: PubMed
    Score: 0.013
  15. Nephron-deficient Fvb mice develop rapidly progressive renal failure and heavy albuminuria involving excess glomerular GLUT1 and VEGF. Lab Invest. 2010 Jan; 90(1):83-97.
    View in: PubMed
    Score: 0.013
  16. Renal FcRn reclaims albumin but facilitates elimination of IgG. J Am Soc Nephrol. 2009 Sep; 20(9):1941-52.
    View in: PubMed
    Score: 0.013
  17. Focal and segmental glomerulosclerosis induced in mice lacking decay-accelerating factor in T cells. J Clin Invest. 2009 May; 119(5):1264-74.
    View in: PubMed
    Score: 0.012
  18. Mouse podocyte complement factor H: the functional analog to human complement receptor 1. J Am Soc Nephrol. 2007 Apr; 18(4):1157-66.
    View in: PubMed
    Score: 0.011
  19. Distinct and separable roles of the complement system in factor H-deficient bone marrow chimeric mice with immune complex disease. J Am Soc Nephrol. 2006 May; 17(5):1354-61.
    View in: PubMed
    Score: 0.010
Connection Strength

The connection strength for concepts is the sum of the scores for each matching publication.

Publication scores are based on many factors, including how long ago they were written and whether the person is a first or senior author.