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Connection

Anthony Chang to Mice

This is a "connection" page, showing publications Anthony Chang has written about Mice.
Connection Strength

0.392
  1. Podocyte injury at young age causes premature senescence and worsens glomerular aging. Am J Physiol Renal Physiol. 2024 01 01; 326(1):F120-F134.
    View in: PubMed
    Score: 0.022
  2. Inhibiting NLRP3 signaling in aging podocytes improves their life- and health-span. Aging (Albany NY). 2023 07 23; 15(14):6658-6689.
    View in: PubMed
    Score: 0.022
  3. Upregulated PD-1 signaling antagonizes glomerular health in aged kidneys and disease. J Clin Invest. 2022 08 15; 132(16).
    View in: PubMed
    Score: 0.020
  4. Macrophage Depletion Reduces Disease Pathology in Factor H-Dependent Immune Complex-Mediated Glomerulonephritis. J Immunol Res. 2022; 2022:1737419.
    View in: PubMed
    Score: 0.020
  5. Innate-like self-reactive B cells infiltrate human renal allografts during transplant rejection. Nat Commun. 2021 07 16; 12(1):4372.
    View in: PubMed
    Score: 0.019
  6. Local complement factor H protects kidney endothelial cell structure and function. Kidney Int. 2021 10; 100(4):824-836.
    View in: PubMed
    Score: 0.019
  7. Exercise alleviates symptoms of CNS lupus. Brain Res. 2021 08 15; 1765:147478.
    View in: PubMed
    Score: 0.019
  8. Differential expression of parietal epithelial cell and podocyte extracellular matrix proteins in focal segmental glomerulosclerosis and diabetic nephropathy. Am J Physiol Renal Physiol. 2019 12 01; 317(6):F1680-F1694.
    View in: PubMed
    Score: 0.017
  9. Quantifying in situ adaptive immune cell cognate interactions in humans. Nat Immunol. 2019 04; 20(4):503-513.
    View in: PubMed
    Score: 0.016
  10. WT1 Is Necessary for the Proliferation and Migration of Cells of Renin Lineage Following Kidney Podocyte Depletion. Stem Cell Reports. 2017 10 10; 9(4):1152-1166.
    View in: PubMed
    Score: 0.015
  11. Bcl-2 as a Therapeutic Target in Human Tubulointerstitial Inflammation. Arthritis Rheumatol. 2016 11; 68(11):2740-2751.
    View in: PubMed
    Score: 0.014
  12. Classifying murine glomerulonephritis using optical coherence tomography and optical coherence elastography. J Biophotonics. 2016 08; 9(8):781-91.
    View in: PubMed
    Score: 0.013
  13. Raman spectroscopy as a diagnostic tool for monitoring acute nephritis. J Biophotonics. 2016 Mar; 9(3):260-9.
    View in: PubMed
    Score: 0.012
  14. Loss of CD11b exacerbates murine complement-mediated tubulointerstitial nephritis. PLoS One. 2014; 9(3):e92051.
    View in: PubMed
    Score: 0.011
  15. Contrasting effects of systemic monocyte/macrophage and CD4+ T cell depletion in a reversible ureteral obstruction mouse model of chronic kidney disease. Clin Dev Immunol. 2013; 2013:836989.
    View in: PubMed
    Score: 0.011
  16. Longitudinal changes in MRI markers in a reversible unilateral ureteral obstruction mouse model: preliminary experience. J Magn Reson Imaging. 2014 Apr; 39(4):835-41.
    View in: PubMed
    Score: 0.011
  17. TNF-mediated damage to glomerular endothelium is an important determinant of acute kidney injury in sepsis. Kidney Int. 2014 Jan; 85(1):72-81.
    View in: PubMed
    Score: 0.011
  18. Curcumin alleviates immune-complex-mediated glomerulonephritis in factor-H-deficient mice. Immunology. 2013 Jul; 139(3):328-37.
    View in: PubMed
    Score: 0.011
  19. Vitamin D receptor signaling in podocytes protects against diabetic nephropathy. J Am Soc Nephrol. 2012 Dec; 23(12):1977-86.
    View in: PubMed
    Score: 0.010
  20. The C5a receptor has a key role in immune complex glomerulonephritis in complement factor H-deficient mice. Kidney Int. 2012 Nov; 82(9):961-8.
    View in: PubMed
    Score: 0.010
  21. Abnormal immune complex processing and spontaneous glomerulonephritis in complement factor H-deficient mice with human complement receptor 1 on erythrocytes. J Immunol. 2010 Sep 15; 185(6):3759-67.
    View in: PubMed
    Score: 0.009
  22. Vitamin D receptor attenuates renal fibrosis by suppressing the renin-angiotensin system. J Am Soc Nephrol. 2010 Jun; 21(6):966-73.
    View in: PubMed
    Score: 0.009
  23. Transgenic overexpression of GLUT1 in mouse glomeruli produces renal disease resembling diabetic glomerulosclerosis. Am J Physiol Renal Physiol. 2010 Jul; 299(1):F99-F111.
    View in: PubMed
    Score: 0.009
  24. Combined vitamin D analog and AT1 receptor antagonist synergistically block the development of kidney disease in a model of type 2 diabetes. Kidney Int. 2010 Jun; 77(11):1000-9.
    View in: PubMed
    Score: 0.009
  25. Chronic kidney disease induced in mice by reversible unilateral ureteral obstruction is dependent on genetic background. Am J Physiol Renal Physiol. 2010 Apr; 298(4):F1024-32.
    View in: PubMed
    Score: 0.009
  26. Nephron-deficient Fvb mice develop rapidly progressive renal failure and heavy albuminuria involving excess glomerular GLUT1 and VEGF. Lab Invest. 2010 Jan; 90(1):83-97.
    View in: PubMed
    Score: 0.008
  27. Renal FcRn reclaims albumin but facilitates elimination of IgG. J Am Soc Nephrol. 2009 Sep; 20(9):1941-52.
    View in: PubMed
    Score: 0.008
  28. Focal and segmental glomerulosclerosis induced in mice lacking decay-accelerating factor in T cells. J Clin Invest. 2009 May; 119(5):1264-74.
    View in: PubMed
    Score: 0.008
  29. Mouse podocyte complement factor H: the functional analog to human complement receptor 1. J Am Soc Nephrol. 2007 Apr; 18(4):1157-66.
    View in: PubMed
    Score: 0.007
  30. Unrestricted C3 activation occurs in Crry-deficient kidneys and rapidly leads to chronic renal failure. J Am Soc Nephrol. 2007 Mar; 18(3):811-22.
    View in: PubMed
    Score: 0.007
  31. Distinct and separable roles of the complement system in factor H-deficient bone marrow chimeric mice with immune complex disease. J Am Soc Nephrol. 2006 May; 17(5):1354-61.
    View in: PubMed
    Score: 0.007
Connection Strength

The connection strength for concepts is the sum of the scores for each matching publication.

Publication scores are based on many factors, including how long ago they were written and whether the person is a first or senior author.