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Connection

Brandon L. Pierce to Polymorphism, Single Nucleotide

This is a "connection" page, showing publications Brandon L. Pierce has written about Polymorphism, Single Nucleotide.
  1. The contribution of parent-to-offspring transmission of telomeres to the heritability of telomere length in humans. Hum Genet. 2019 Jan; 138(1):49-60.
    View in: PubMed
    Score: 0.368
  2. The Genetic Architecture of Arsenic Metabolism Efficiency:A SNP-Based Heritability Study of Bangladeshi Adults. Environ Health Perspect. 2015 10; 123(10):985-92.
    View in: PubMed
    Score: 0.284
  3. Genome-wide association study identifies chromosome 10q24.32 variants associated with arsenic metabolism and toxicity phenotypes in Bangladesh. PLoS Genet. 2012; 8(2):e1002522.
    View in: PubMed
    Score: 0.230
  4. Association study of type 2 diabetes genetic susceptibility variants and risk of pancreatic cancer: an analysis of PanScan-I data. Cancer Causes Control. 2011 Jun; 22(6):877-83.
    View in: PubMed
    Score: 0.216
  5. Clinical assessment incorporating a personal genome. Lancet. 2010 Sep 11; 376(9744):869; author reply 869-70.
    View in: PubMed
    Score: 0.208
  6. The Impact of Inherited Genetic Variation on DNA Methylation in Prostate Cancer and Benign Tissues of African American and European American Men. Cancer Epidemiol Biomarkers Prev. 2024 Apr 03; 33(4):557-566.
    View in: PubMed
    Score: 0.133
  7. Sequencing-based fine-mapping and in silico functional characterization of the 10q24.32 arsenic metabolism efficiency locus across multiple arsenic-exposed populations. PLoS Genet. 2023 01; 19(1):e1010588.
    View in: PubMed
    Score: 0.122
  8. Rare, Protein-Altering Variants in AS3MT and Arsenic Metabolism Efficiency: A Multi-Population Association Study. Environ Health Perspect. 2021 04; 129(4):47007.
    View in: PubMed
    Score: 0.108
  9. Genetic Determinants of Reduced Arsenic Metabolism Efficiency in the 10q24.32 Region Are Associated With Reduced AS3MT Expression in Multiple Human Tissue Types. Toxicol Sci. 2020 08 01; 176(2):382-395.
    View in: PubMed
    Score: 0.103
  10. A missense variant in FTCD is associated with arsenic metabolism and toxicity phenotypes in Bangladesh. PLoS Genet. 2019 03; 15(3):e1007984.
    View in: PubMed
    Score: 0.094
  11. Co-occurring expression and methylation QTLs allow detection of common causal variants and shared biological mechanisms. Nat Commun. 2018 02 23; 9(1):804.
    View in: PubMed
    Score: 0.087
  12. Screening for gene-environment (G×E) interaction using omics data from exposed individuals: an application to gene-arsenic interaction. Mamm Genome. 2018 02; 29(1-2):101-111.
    View in: PubMed
    Score: 0.087
  13. Genome-wide association study of telomere length among South Asians identifies a second RTEL1 association signal. J Med Genet. 2018 01; 55(1):64-71.
    View in: PubMed
    Score: 0.086
  14. Mendelian randomization study of adiposity-related traits and risk of breast, ovarian, prostate, lung and colorectal cancer. Int J Epidemiol. 2016 06; 45(3):896-908.
    View in: PubMed
    Score: 0.078
  15. A unified set-based test with adaptive filtering for gene-environment interaction analyses. Biometrics. 2016 06; 72(2):629-38.
    View in: PubMed
    Score: 0.074
  16. Genome-wide association study of parity in Bangladeshi women. PLoS One. 2015; 10(3):e0118488.
    View in: PubMed
    Score: 0.071
  17. Genome-wide association study of smoking behaviours among Bangladeshi adults. J Med Genet. 2014 May; 51(5):327-33.
    View in: PubMed
    Score: 0.066
  18. Genetic susceptibility to accelerated cognitive decline in the US Health and Retirement Study. Neurobiol Aging. 2014 Jun; 35(6):1512.e11-8.
    View in: PubMed
    Score: 0.065
  19. Arsenic metabolism efficiency has a causal role in arsenic toxicity: Mendelian randomization and gene-environment interaction. Int J Epidemiol. 2013 Dec; 42(6):1862-71.
    View in: PubMed
    Score: 0.065
  20. Unidentified genetic variants influence pancreatic cancer risk: an analysis of polygenic susceptibility in the PanScan study. Genet Epidemiol. 2012 Jul; 36(5):517-24.
    View in: PubMed
    Score: 0.059
  21. Genome-wide "pleiotropy scan" identifies HNF1A region as a novel pancreatic cancer susceptibility locus. Cancer Res. 2011 Jul 01; 71(13):4352-8.
    View in: PubMed
    Score: 0.054
  22. Genetic susceptibility to type 2 diabetes is associated with reduced prostate cancer risk. Hum Hered. 2010; 69(3):193-201.
    View in: PubMed
    Score: 0.050
  23. C-reactive protein, interleukin-6, and prostate cancer risk in men aged 65 years and older. Cancer Causes Control. 2009 Sep; 20(7):1193-203.
    View in: PubMed
    Score: 0.047
  24. Genomics of 1 million parent lifespans implicates novel pathways and common diseases and distinguishes survival chances. Elife. 2019 01 15; 8.
    View in: PubMed
    Score: 0.046
  25. Integrative cross-omics and cross-context analysis elucidates molecular links underlying genetic effects on complex traits. Nat Commun. 2024 Mar 16; 15(1):2383.
    View in: PubMed
    Score: 0.033
  26. Large-scale cis- and trans-eQTL analyses identify thousands of genetic loci and polygenic scores that regulate blood gene expression. Nat Genet. 2021 09; 53(9):1300-1310.
    View in: PubMed
    Score: 0.028
  27. Primo: integration of multiple GWAS and omics QTL summary statistics for elucidation of molecular mechanisms of trait-associated SNPs and detection of pleiotropy in complex traits. Genome Biol. 2020 09 11; 21(1):236.
    View in: PubMed
    Score: 0.026
  28. Circulating vitamin D concentrations and risk of breast and prostate cancer: a Mendelian randomization study. Int J Epidemiol. 2019 10 01; 48(5):1416-1424.
    View in: PubMed
    Score: 0.024
  29. Circulating vitamin D concentration and risk of seven cancers: Mendelian randomisation study. BMJ. 2017 10 31; 359:j4761.
    View in: PubMed
    Score: 0.021
  30. Identifying cis-mediators for trans-eQTLs across many human tissues using genomic mediation analysis. Genome Res. 2017 11; 27(11):1859-1871.
    View in: PubMed
    Score: 0.021
  31. Trans-ethnic predicted expression genome-wide association analysis identifies a gene for estrogen receptor-negative breast cancer. PLoS Genet. 2017 Sep; 13(9):e1006727.
    View in: PubMed
    Score: 0.021
  32. Telomere structure and maintenance gene variants and risk of five cancer types. Int J Cancer. 2016 Dec 15; 139(12):2655-2670.
    View in: PubMed
    Score: 0.020
  33. Determinants and Consequences of Arsenic Metabolism Efficiency among 4,794 Individuals: Demographics, Lifestyle, Genetics, and Toxicity. Cancer Epidemiol Biomarkers Prev. 2016 Feb; 25(2):381-90.
    View in: PubMed
    Score: 0.019
  34. Genetic determinants of telomere length and risk of common cancers: a Mendelian randomization study. Hum Mol Genet. 2015 Sep 15; 24(18):5356-66.
    View in: PubMed
    Score: 0.018
  35. Interaction between arsenic exposure from drinking water and genetic polymorphisms on cardiovascular disease in Bangladesh: a prospective case-cohort study. Environ Health Perspect. 2015 May; 123(5):451-7.
    View in: PubMed
    Score: 0.018
  36. A genome-wide association study of early-onset breast cancer identifies PFKM as a novel breast cancer gene and supports a common genetic spectrum for breast cancer at any age. Cancer Epidemiol Biomarkers Prev. 2014 Apr; 23(4):658-69.
    View in: PubMed
    Score: 0.016
  37. Localization of PSORS1 to a haplotype block harboring HLA-C and distinct from corneodesmosin and HCR. Hum Genet. 2005 Dec; 118(3-4):466-76.
    View in: PubMed
    Score: 0.009
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