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Froelich, Christopher
One or more keywords matched the following items that are connected to Froelich, Christopher
Item TypeName
Concept Caspases
Academic Article ICE-LAP6, a novel member of the ICE/Ced-3 gene family, is activated by the cytotoxic T cell protease granzyme B.
Academic Article Granzyme B mimics apical caspases. Description of a unified pathway for trans-activation of executioner caspase-3 and -7.
Academic Article Cytotoxic T-cell-derived granzyme B activates the apoptotic protease ICE-LAP3.
Academic Article Pro-caspase-3 is a major physiologic target of caspase-8.
Academic Article Cell death-associated translocation of plasma membrane components induced by CTL.
Academic Article Reconstitution of caspase 3 sensitizes MCF-7 breast cancer cells to doxorubicin- and etoposide-induced apoptosis.
Academic Article Granzyme B-mediated apoptosis--the elephant and the blind men?
Academic Article Reconstitution of caspase-3 confers low glucose-enhanced tumor necrosis factor-related apoptosis-inducing ligand cytotoxicity and Akt cleavage.
Academic Article Perforin and granzyme B induce apoptosis in FasL-resistant colon carcinoma cells.
Academic Article The T cell death knell: immune-mediated tumor death in renal cell carcinoma.
Academic Article Granzyme B activates procaspase-3 which signals a mitochondrial amplification loop for maximal apoptosis.
Academic Article Granzyme A is a proinflammatory protease.
Academic Article NK cell protease granzyme M targets alpha-tubulin and disorganizes the microtubule network.
Academic Article Granule-mediated killing: pathways for granzyme B-initiated apoptosis.
Academic Article FLIP prevents apoptosis induced by death receptors but not by perforin/granzyme B, chemotherapeutic drugs, and gamma irradiation.
Academic Article Apoptosis induced by granzyme B-glycosaminoglycan complexes: implications for granule-mediated apoptosis in vivo.
Academic Article Degradation of Mcl-1 by granzyme B: implications for Bim-mediated mitochondrial apoptotic events.
Academic Article Mcl-1 is a key regulator of apoptosis resistance in Chlamydia trachomatis-infected cells.
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