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Wickrema, Amittha
One or more keywords matched the following items that are connected to Wickrema, Amittha
Item TypeName
Concept Myelodysplastic Syndromes
Academic Article Reduced SMAD7 leads to overactivation of TGF-beta signaling in MDS that can be reversed by a specific inhibitor of TGF-beta receptor I kinase.
Academic Article Aberrant epigenetic and genetic marks are seen in myelodysplastic leukocytes and reveal Dock4 as a candidate pathogenic gene on chromosome 7q.
Academic Article Fludarabine, melphalan, and alemtuzumab conditioning in adults with standard-risk advanced acute myeloid leukemia and myelodysplastic syndrome.
Academic Article Inhibition of the TGF-beta receptor I kinase promotes hematopoiesis in MDS.
Academic Article Identifying familial myelodysplastic/acute leukemia predisposition syndromes through hematopoietic stem cell transplantation donors with thrombocytopenia.
Academic Article IL8-CXCR2 pathway inhibition as a therapeutic strategy against MDS and AML stem cells.
Academic Article Quantitative characterization of T-cell repertoire in allogeneic hematopoietic stem cell transplant recipients.
Academic Article Reduced DOCK4 expression leads to erythroid dysplasia in myelodysplastic syndromes.
Academic Article The prognostic value of serum C-reactive protein, ferritin, and albumin prior to allogeneic transplantation for acute myeloid leukemia and myelodysplastic syndromes.
Academic Article Identifying Inherited and Acquired Genetic Factors Involved in Poor Stem Cell Mobilization and Donor-Derived Malignancy.
Academic Article Epigenetically Aberrant Stroma in MDS Propagates Disease via Wnt/ß-Catenin Activation.
Academic Article Antisense STAT3 inhibitor decreases viability of myelodysplastic and leukemic stem cells.
Academic Article Reduced-Intensity Allogeneic Transplant for Acute Myeloid Leukemia and Myelodysplastic Syndrome Using Combined CD34-Selected Haploidentical Graft and a Single Umbilical Cord Unit Compared with Matched Unrelated Donor Stem Cells in Older Adults.
Academic Article Disruption of erythroid nuclear opening and histone release in myelodysplastic syndromes.
Academic Article Loss of Function of DOCK4 in Myelodysplastic Syndromes Stem Cells is Restored by Inhibitors of DOCK4 Signaling Networks.
Academic Article Activation of targetable inflammatory immune signaling is seen in myelodysplastic syndromes with SF3B1 mutations.
Grant The role of Dock4 in normal and aberrant erythropoiesis
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  • Myelodysplastic Syndromes