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Monoclonal anti-acetylcholine receptor antibodies can cause experimental myasthenia.Academic Article Why?
The beta 2-adrenergic agonist terbutaline suppresses acute passive transfer experimental autoimmune myasthenia gravis (EAMG).Academic Article Why?
Myasthenia Gravis, Autoimmune, ExperimentalConcept Why?
Anti-acetylcholine receptor antibodies directed against the alpha-bungarotoxin binding site induce a unique form of experimental myasthenia.Academic Article Why?
Induction of the morphologic changes of both acute and chronic experimental myasthenia by monoclonal antibody directed against acetylcholine receptor.Academic Article Why?
Refractoriness to a second episode of experimental myasthenia gravis. Correlation with AChR concentration and morphologic appearance of the postsynaptic membrane.Academic Article Why?
Use of monoclonal antiacetylcholine receptor antibodies to investigate the macrophage inflammation of acute experimental myasthenia gravis: refractoriness to a second episode of acute disease.Academic Article Why?
Animal models of antimuscle-specific kinase myasthenia.Academic Article Why?
CD1d(hi)CD5+ B cells expanded by GM-CSF in vivo suppress experimental autoimmune myasthenia gravis.Academic Article Why?
IL-10 derived from CD1dhiCD5? B cells regulates experimental autoimmune myasthenia gravis.Academic Article Why?
Myeloid-derived suppressor cells as a potential therapy for experimental autoimmune myasthenia gravis.Academic Article Why?
Soliven, BettyPerson Why?
Fung, JohnPerson Why?
Pytel, PeterPerson Why?
Arnason, Barry G.Person Why?
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