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Kron, StephenPerson Why?
Lipid-derived reactive aldehydes link oxidative stress to cell senescence.Academic Article Why?
PAI-1 contributes to homocysteine-induced cellular senescence.Academic Article Why?
ESE-3, an Ets family transcription factor, is up-regulated in cellular senescence.Academic Article Why?
Overcoming cellular senescence in human cancer pathogenesis.Academic Article Why?
A cmap-enabled gene expression signature-matching approach identifies small-molecule inducers of accelerated cell senescence.Academic Article Why?
Therapy-Induced Cellular Senescence: Potentiating Tumor Elimination or Driving Cancer Resistance and Recurrence?Academic Article Why?
Transforming malignant tumors into vulnerable phenotypes via nanoscale coordination polymer mediated cell senescence and photodynamic therapy.Academic Article Why?
Targets of Reactive Lipid Species regulating DNA damage response and cell senescenceGrant Why?
A small molecule inhibitor of PAI-1 protects against doxorubicin-induced cellular senescence.Academic Article Why?
Long-term male-specific chronic pain via telomere- and p53-mediated spinal cord cellular senescence.Academic Article Why?
p52 signaling promotes cellular senescence.Academic Article Why?
Cellular SenescenceConcept Why?
Lipid signaling in cellular senescence and tissue agingGrant Why?
Resequencing Study Confirms That Host Defense and Cell Senescence Gene Variants Contribute to the Risk of Idiopathic Pulmonary Fibrosis.Academic Article Why?
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