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MatchTypeWhy
Staley, Jonathan P.Person Why?
Multiple functions for the invariant AGC triad of U6 snRNA.Academic Article Why?
Human DNA sequences complementary to the small nuclear RNA U2.Academic Article Why?
RNA, Small NuclearConcept Why?
Specific alterations of U1-C protein or U1 small nuclear RNA can eliminate the requirement of Prp28p, an essential DEAD box splicing factor.Academic Article Why?
Termination of pre-mRNA splicing requires that the ATPase and RNA unwindase Prp43p acts on the catalytic snRNA U6.Academic Article Why?
The highly conserved Chinese hamster GNAI3 gene maps less than 60 kb from the AMPD2 gene and lacks the intronic U6 snRNA present in its human counterpart.Academic Article Why?
Biallelic mutations in the 3' exonuclease TOE1 cause pontocerebellar hypoplasia and uncover a role in snRNA processing.Academic Article Why?
Altered expression level of a systemic nuclear autoantigen determines the fate of immune response to self.Academic Article Why?
Clinical value of an integrated ELISA system for the detection of 6 autoantibodies (ssDNA, dsDNA, Sm, RNP/Sm, SSA, and SSB).Academic Article Why?
Cloning of the human cDNA for the U1 RNA-associated 70K protein.Academic Article Why?
Evidence that U2/U6 helix I promotes both catalytic steps of pre-mRNA splicing and rearranges in between these steps.Academic Article Why?
Formation of the 3' end on U snRNAs requires at least three sequence elements.Academic Article Why?
Properties of a U1 RNA enhancer-like sequence.Academic Article Why?
RNA catalyses nuclear pre-mRNA splicing.Academic Article Why?
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