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Search Results to Yu-Ying He

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One or more keywords matched the following properties of He, Yu-Ying

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keywords Autophagy
overview Our research addresses the fundamental questions: how normal cells respond to environmental carcinogens and evolve into cancerous cells. In particular, our group focuses on the role of epitranscriptomics, epigenetics, and autophagy in regulating genomic integrity and cellular homeostasis and to investigate their impact on cancer development and therapeutic resistance, with an emphasis on skin cancer including squamous cancer and melanoma. Our long-term goal is to identify previously unrecognized, therapeutically accessible molecular regulatory networks that predict susceptibility to skin cancer, and improve our ability to prevent and treat these cancers.

One or more keywords matched the following items that are connected to He, Yu-Ying

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Academic Article Autophagy controls p38 activation to promote cell survival under genotoxic stress.
Academic Article Autophagy deficiency stabilizes TWIST1 to promote epithelial-mesenchymal transition.
Academic Article Autophagy positively regulates DNA damage recognition by nucleotide excision repair.
Academic Article Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition).
Academic Article Autophagy in UV Damage Response.
Academic Article NF-?B Signaling Activation Induced by Chloroquine Requires Autophagosome, p62 Protein, and c-Jun N-terminal Kinase (JNK) Signaling and Promotes Tumor Cell Resistance.
Academic Article Autophagy gene ATG7 regulates ultraviolet radiation-induced inflammation and skin tumorigenesis.
Academic Article Mitochondrial dysfunction activates the AMPK signaling and autophagy to promote cell survival.
Academic Article Mechanisms and prevention of UV-induced melanoma.
Academic Article The Autophagy Receptor Adaptor p62 is Up-regulated by UVA Radiation in Melanocytes and in Melanoma Cells.
Academic Article Arsenic Induces p62 Expression to Form a Positive Feedback Loop with Nrf2 in Human Epidermal Keratinocytes: Implications for Preventing Arsenic-Induced Skin Cancer.
Academic Article Keratinocyte autophagy enables the activation of keratinocytes and fibroblastsand facilitates wound healing.
Academic Article Autophagy of the m6A mRNA demethylase FTO is impaired by low-level arsenic exposure to promote tumorigenesis.

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  • Autophagy