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Addition of endothelial progenitor cells to renal revascularization restores medullary tubular oxygen consumption in swine renal artery stenosis.
Changes in glomerular filtration rate after renal revascularization correlate with microvascular hemodynamics and inflammation in Swine renal artery stenosis.
Mitochondrial targeted peptides attenuate residual myocardial damage after reversal of experimental renovascular hypertension.
Restoration of Mitochondrial Cardiolipin Attenuates Cardiac Damage in Swine Renovascular Hypertension.
Early atherosclerosis aggravates renal microvascular loss and fibrosis in swine renal artery stenosis.