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bcl-2-Associated X Protein
BAX and BAK regulation of endoplasmic reticulum Ca2+: a control point for apoptosis.
Regulation of endoplasmic reticulum Ca2+ dynamics by proapoptotic BCL-2 family members.
Proapoptotic BAX and BAK regulate the type 1 inositol trisphosphate receptor and calcium leak from the endoplasmic reticulum.
A distinct pathway remodels mitochondrial cristae and mobilizes cytochrome c during apoptosis.
Caspase-2 cleavage of BID is a critical apoptotic signal downstream of endoplasmic reticulum stress.
Blocking the mitochondrial apoptotic pathway preserves motor neuron viability and function in a mouse model of amyotrophic lateral sclerosis.
bcl 2 Associated X Protein