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Platelet-type 12-lipoxygenase deletion provokes a compensatory 12/15-lipoxygenase increase that exacerbates oxidative stress in mouse islet ß cells.
12-lipoxygenase promotes obesity-induced oxidative stress in pancreatic islets.
Inhibition of 12/15-Lipoxygenase Protects Against ß-Cell Oxidative Stress and Glycemic Deterioration in Mouse Models of Type 1 Diabetes.
Molecular mechanisms of nonalcoholic fatty liver disease: Potential role for 12-lipoxygenase.
Transcriptional and Translational Mechanisms Governing Beta Cell Function
Role of 12-lipoxygenase and 12-HETE signaling in beta-cell dysfunction
The 12-HETE receptor Gpr31 in the -cell pathogenesis of type 1 diabetes
Inside the ß Cell: Molecular Stress Response Pathways in Diabetes Pathogenesis.