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Monocyte chemoattractant protein-1 (MCP-1) and macrophage infiltration into the skin after burn injury in aged mice.
Immunosuppression after injury in aged mice is associated with a TH1-TH2 shift, which can be restored by estrogen treatment.
Aging enhances lymphocyte cytokine defects after injury.
Advanced age negatively influences mesenteric lymph node T cell responses after burn injury.
Lack of aromatase improves cell-mediated immune response after burn.
The early acute phase response after burn injury in mice.
Neutrophil Accumulation in the Small Intestine Contributes to Local Tissue Destruction Following Combined Radiation and Burn Injury.
Effects of Estrogen on Bacterial Clearance and Neutrophil Response After Combined Burn Injury and Wound Infection.
Estrogen replacement, aging, and cell-mediated immunity after injury.
A low blood ethanol level is associated with improved cytokine production in aged mice after traumatic injury.
Aging and estrogen: modulation of inflammatory responses after injury.
Gender-based differences in cytokine production after burn injury: a role of interleukin-6.
Survival and cell mediated immunity after burn injury in aged mice.