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Search Results to Christopher M. Gomez

This is a "connection" page, showing the details of why an item matched the keywords from your search.

                     
                     

One or more keywords matched the following items that are connected to Gomez, Christopher M.

Item TypeName
Concept Receptors, Cholinergic
Academic Article A transgenic mouse model of the slow-channel syndrome.
Academic Article Slow-channel transgenic mice: a model of postsynaptic organellar degeneration at the neuromuscular junction.
Academic Article Genetic manipulation of AChR responses suggests multiple causes of weakness in slow-channel syndrome.
Academic Article Active calcium accumulation underlies severe weakness in a panel of mice with slow-channel syndrome.
Academic Article Focal caspase activation underlies the endplate myopathy in slow-channel syndrome.
Academic Article Activation of apoptotic pathways at muscle fiber synapses is circumscribed and reversible in a slow-channel syndrome model.
Academic Article Induction of the morphologic changes of both acute and chronic experimental myasthenia by monoclonal antibody directed against acetylcholine receptor.
Academic Article Monoclonal hybridoma anti-acetylcholine receptor antibodies: antibody specificity and effect of passive transfer.
Academic Article Skeletal muscle IP3R1 receptors amplify physiological and pathological synaptic calcium signals.
Academic Article Use of monoclonal antiacetylcholine receptor antibodies to investigate the macrophage inflammation of acute experimental myasthenia gravis: refractoriness to a second episode of acute disease.
Academic Article Monoclonal anti-acetylcholine receptor antibodies can cause experimental myasthenia.
Academic Article Desensitization of mutant acetylcholine receptors in transgenic mice reduces the amplitude of neuromuscular synaptic currents.
Academic Article Monoclonal anti-acetylcholine receptor antibodies with differing capacities to induce experimental autoimmune myasthenia gravis.
Academic Article A beta-subunit mutation in the acetylcholine receptor channel gate causes severe slow-channel syndrome.
Academic Article A leucine-to-phenylalanine substitution in the acetylcholine receptor ion channel in a family with the slow-channel syndrome.
Academic Article Inositol-1,4,5-triphosphate receptors mediate activity-induced synaptic Ca2+ signals in muscle fibers and Ca2+ overload in slow-channel syndrome.
Academic Article Macroscopic properties of spontaneous mutations in slow-channel syndrome: correlation by domain and disease severity.
Academic Article Myasthenia induced by monoclonal anti-acetylcholine receptor antibodies: clinical and electrophysiological aspects.
Academic Article Anti-acetylcholine receptor antibodies directed against the alpha-bungarotoxin binding site induce a unique form of experimental myasthenia.

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  • Receptors Cholinergic