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Prabhakar, Nanduri R.
One or more keywords matched the following items that are connected to Prabhakar, Nanduri R.
Item TypeName
Concept Calcium Channel Agonists
Concept Calcium Channels
Concept Calcium Signaling
Concept Calcium Channels, T-Type
Concept Calcium Channels, N-Type
Concept Ryanodine Receptor Calcium Release Channel
Concept Calcium-Calmodulin-Dependent Protein Kinase Type 2
Concept Calcium-Calmodulin-Dependent Protein Kinases
Concept Calcium
Concept Calcium Channel Blockers
Concept Calcium-Binding Proteins
Concept Calcium Channels, L-Type
Academic Article Heterogeneity in cytosolic calcium responses to hypoxia in carotid body cells.
Academic Article Carbon monoxide and carotid body chemoreception.
Academic Article Norepinephrine inhibits a toxin resistant Ca2+ current in carotid body glomus cells: evidence for a direct G protein mechanism.
Academic Article Ca2+ current in rabbit carotid body glomus cells is conducted by multiple types of high-voltage-activated Ca2+ channels.
Academic Article Intracellular pathways linking hypoxia to activation of c-fos and AP-1.
Academic Article Release of dopamine and norepinephrine by hypoxia from PC-12 cells.
Academic Article Nitric oxide inhibits L-type Ca2+ current in glomus cells of the rabbit carotid body via a cGMP-independent mechanism.
Academic Article HERG-Like potassium current regulates the resting membrane potential in glomus cells of the rabbit carotid body.
Academic Article CO2/HCO3- modulates K+ and Ca2+ currents in glomus cells of the carotid body.
Academic Article Augmentation of L-type calcium current by hypoxia in rabbit carotid body glomus cells: evidence for a PKC-sensitive pathway.
Academic Article Intermittent hypoxia: cell to system.
Academic Article Release of substance P by low oxygen in the rabbit carotid body: evidence for the involvement of calcium channels.
Academic Article CO(2) and pH independently modulate L-type Ca(2+) current in rabbit carotid body glomus cells.
Academic Article Secretion of brain-derived neurotrophic factor from PC12 cells in response to oxidative stress requires autocrine dopamine signaling.
Academic Article Induction of HIF-1alpha expression by intermittent hypoxia: involvement of NADPH oxidase, Ca2+ signaling, prolyl hydroxylases, and mTOR.
Academic Article Intermittent hypoxia degrades HIF-2alpha via calpains resulting in oxidative stress: implications for recurrent apnea-induced morbidities.
Academic Article Neonatal intermittent hypoxia leads to long-lasting facilitation of acute hypoxia-evoked catecholamine secretion from rat chromaffin cells.
Academic Article NADPH oxidase-dependent regulation of T-type Ca2+ channels and ryanodine receptors mediate the augmented exocytosis of catecholamines from intermittent hypoxia-treated neonatal rat chromaffin cells.
Academic Article Hydrogen sulfide (H(2)S): a physiologic mediator of carotid body response to hypoxia.
Academic Article Carbon monoxide (CO) and hydrogen sulfide (H(2)S) in hypoxic sensing by the carotid body.
Academic Article Endogenous H2S is required for hypoxic sensing by carotid body glomus cells.
Academic Article L-type Ca(2+) channel activation regulates induction of c-fos transcription by hypoxia.
Academic Article Chemosensing at the carotid body. Involvement of a HERG-like potassium current in glomus cells.
Academic Article Augmentation of calcium current by hypoxia in carotid body glomus cells.
Academic Article Activation of tyrosine hydroxylase by intermittent hypoxia: involvement of serine phosphorylation.
Academic Article Facilitation of dopamine and acetylcholine release by intermittent hypoxia in PC12 cells: involvement of calcium and reactive oxygen species.
Academic Article Ca2+/calmodulin kinase-dependent activation of hypoxia inducible factor 1 transcriptional activity in cells subjected to intermittent hypoxia.
Academic Article Neonatal intermittent hypoxia impairs neuronal nicotinic receptor expression and function in adrenal chromaffin cells.
Academic Article Regulation of hypoxia-inducible factor-a isoforms and redox state by carotid body neural activity in rats.
Academic Article CaV3.2 T-type Ca²? channels in H2S-mediated hypoxic response of the carotid body.
Academic Article Protein kinase G-regulated production of H2S governs oxygen sensing.
Academic Article CaV3.2 T-type Ca2+ channels mediate the augmented calcium influx in carotid body glomus cells by chronic intermittent hypoxia.
Academic Article H2S mediates carotid body response to hypoxia but not anoxia.
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  • Calcium