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Fehon, Richard G.
One or more keywords matched the following items that are connected to Fehon, Richard G.
Item TypeName
Concept Neurofibromin 2
Academic Article Isolation of mutations in the Drosophila homologues of the human Neurofibromatosis 2 and yeast CDC42 genes using a simple and efficient reverse-genetic method.
Academic Article Distinct cellular and subcellular patterns of expression imply distinct functions for the Drosophila homologues of moesin and the neurofibromatosis 2 tumor suppressor, merlin.
Academic Article Advances in neurofibromatosis 2 (NF2): a workshop report.
Academic Article A systematic screen for dominant second-site modifiers of Merlin/NF2 phenotypes reveals an interaction with blistered/DSRF and scribbler.
Academic Article Structural analysis of Drosophila merlin reveals functional domains important for growth control and subcellular localization.
Academic Article The neurofibromatosis-2 homologue, Merlin, and the tumor suppressor expanded function together in Drosophila to regulate cell proliferation and differentiation.
Academic Article ERM proteins and merlin: integrators at the cell cortex.
Academic Article Self-masking in an intact ERM-merlin protein: an active role for the central alpha-helical domain.
Academic Article Microtubule-mediated transport of the tumor-suppressor protein Merlin and its mutants.
Academic Article Phosphorylation and activity of the tumor suppressor Merlin and the ERM protein Moesin are coordinately regulated by the Slik kinase.
Academic Article The tumor suppressors Merlin and Expanded function cooperatively to modulate receptor endocytosis and signaling.
Academic Article Merlin and the ERM proteins--regulators of receptor distribution and signaling at the cell cortex.
Academic Article In vivo functional analysis of the human NF2 tumor suppressor gene in Drosophila.
Academic Article Kibra and Merlin Activate the Hippo Pathway Spatially Distinct from and Independent of Expanded.
Academic Article Negative feedback couples Hippo pathway activation with Kibra degradation independent of Yorkie-mediated transcription.
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  • Neurofibromin 2