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Histocompatibility Antigens Class II
Histocompatibility Antigens Class I
Antigenic cancer cells grow progressively in immune hosts without evidence for T cell exhaustion or systemic anergy.
Immunodominance deters the response to other tumor antigens thereby favoring escape: prevention by vaccination with tumor variants selected with cloned cytolytic T cells in vitro.
Complementary role of CD4+ T cells and secondary lymphoid tissues for cross-presentation of tumor antigen to CD8+ T cells.
Long-term persistence of CD4(+) but rapid disappearance of CD8(+) T cells expressing an MHC class I-restricted TCR of nanomolar affinity.
Highly immunogenic regressor tumor cells can prevent development of postsurgical tumor immunity.
Animals bearing malignant grafts reject normal grafts that express through gene transfer the same antigen.
CD4(+) T cells eliminate MHC class II-negative cancer cells in vivo by indirect effects of IFN-gamma.
Immunodominance and tumor escape.
MHC-class I-restricted CD4 T cells: a nanomolar affinity TCR has improved anti-tumor efficacy in vivo compared to the micromolar wild-type TCR.
Targeting cancer-specific mutations by T cell receptor gene therapy.
Transfer of Allogeneic CD4+ T Cells Rescues CD8+ T Cells in Anti-PD-L1-Resistant Tumors Leading to Tumor Eradication.
TCR-pMHC bond conformation controls TCR ligand discrimination.
Histocompatibility Antigen H-2D
MANIPULATION OF TUMOR-SPECIFIC IMMUNITY