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Rad51 is an accessory factor for Dmc1-mediated joint molecule formation during meiosis.
RI-1: a chemical inhibitor of RAD51 that disrupts homologous recombination in human cells.
An optimized RAD51 inhibitor that disrupts homologous recombination without requiring Michael acceptor reactivity.
Real-time solution measurement of RAD51- and RecA-mediated strand assimilation without background annealing.
The RAD51-stimulatory compound RS-1 can exploit the RAD51 overexpression that exists in cancer cells and tumors.
DNA repair pathway gene expression score correlates with repair proficiency and tumor sensitivity to chemotherapy.
Noncanonical NF-?B factor p100/p52 regulates homologous recombination and modulates sensitivity to DNA-damaging therapy.