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Caenorhabditis elegans Proteins
Prevention of siderophore- mediated gut-derived sepsis due to P. aeruginosa can be achieved without iron provision by maintaining local phosphate abundance: role of pH.
Pseudomonas aeruginosa overrides the virulence inducing effect of opioids when it senses an abundance of phosphate.
Intestinal tissues induce an SNP mutation in Pseudomonas aeruginosa that enhances its virulence: possible role in anastomotic leak.
Host stress and virulence expression in intestinal pathogens: development of therapeutic strategies using mice and C. elegans.
Candida albicans isolates from the gut of critically ill patients respond to phosphate limitation by expressing filaments and a lethal phenotype.
Red death in Caenorhabditis elegans caused by Pseudomonas aeruginosa PAO1.
Membership and behavior of ultra-low-diversity pathogen communities present in the gut of humans during prolonged critical illness.
Collagen degradation and MMP9 activation by Enterococcus faecalis contribute to intestinal anastomotic leak.
Emergence of the P2 phenotype in Pseudomonas aeruginosa PAO1 strains involves various mutations in mexT or mexF.
C. elegans and mutants with chronic nicotine exposure as a novel model of cancer phenotype.