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Advanced glycation of beta 2-microglobulin in the pathogenesis of bone lesions in dialysis-associated amyloidosis.
Serum amyloid P component: a predictor of clinical beta 2-microglobulin amyloidosis.
The pathogenesis of beta(2)-microglobulin-induced bone lesions in dialysis-related amyloidosis.
Assessment of the stability of an immunoadsorbent for the extracorporeal removal of Beta-2-microglobulin from blood.
Beta2-microglobulin stimulates osteoclast formation.
Is beta 2-microglobulin a mediator of bone disease?
Possible involvement of advanced glycation end-products in bone resorption.