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Na(+)-Ca(2+) exchange current and submembrane [Ca(2+)] during the cardiac action potential.
Claudin-2-dependent paracellular channels are dynamically gated.
Cardiac submembrane [Na+] transients sensed by Na+-Ca2+ exchange current.
Dynamic modeling of the tight junction pore pathway.
Intracellular Na(+) concentration is elevated in heart failure but Na/K pump function is unchanged.
Cellular basis of abnormal calcium transients of failing human ventricular myocytes.
Mechanisms and pathways of trans-tight junction conductance
Patch Clamp Techniques