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Molecular Mechanism of the Association Between Atrial Fibrillation and Heart Failure Includes Energy Metabolic Dysregulation Due to Mitochondrial Dysfunction.
Potassium channel openers protect cardiac mitochondria by attenuating oxidant stress at reoxygenation.
Targeting nucleotide-requiring enzymes: implications for diazoxide-induced cardioprotection.
Effective pharmacotherapy against oxidative injury: alternative utility of an ATP-sensitive potassium channel opener.
Mitochondrial Mechanisms and Novel Therapeutic Targets in Atrial Fibrillation