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Increased expression of alternatively spliced dominant-negative isoform of SRF in human failing hearts.
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Increased expression of alternatively spliced dominant-negative isoform of SRF in human failing hearts.
Increased expression of alternatively spliced dominant-negative isoform of SRF in human failing hearts. Am J Physiol Heart Circ Physiol. 2002 Apr; 282(4):H1521-33.
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PubMed
subject areas
Adult
Aged
Aged, 80 and over
Alternative Splicing
Amino Acid Sequence
Animals
Base Sequence
Cell Division
Cells, Cultured
Child
Cloning, Molecular
Disease Models, Animal
DNA Primers
DNA Probes
Exons
Female
Gene Expression Regulation
Heart Failure
Heart Valve Prosthesis Implantation
Humans
Male
Middle Aged
Molecular Sequence Data
Myocardium
Protein Isoforms
Rabbits
Rats
Reference Values
Reverse Transcriptase Polymerase Chain Reaction
Serum Response Factor
Transcription, Genetic
Transcriptional Activation
Transfection
authors with profiles
Mahesh P. Gupta