"Salmeterol Xinafoate" is a descriptor in the National Library of Medicine's controlled vocabulary thesaurus,
MeSH (Medical Subject Headings). Descriptors are arranged in a hierarchical structure,
which enables searching at various levels of specificity.
A selective ADRENERGIC BETA-2 RECEPTOR agonist that functions as a BRONCHODILATOR when administered by inhalation. It is used to manage the symptoms of ASTHMA and CHRONIC OBSTRUCTIVE PULMONARY DISEASE.
Below are MeSH descriptors whose meaning is more general than "Salmeterol Xinafoate".
Below are MeSH descriptors whose meaning is more specific than "Salmeterol Xinafoate".
This graph shows the total number of publications written about "Salmeterol Xinafoate" by people in this website by year, and whether "Salmeterol Xinafoate" was a major or minor topic of these publications.
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Below are the most recent publications written about "Salmeterol Xinafoate" by people in Profiles.
Pharmacogenetic studies of long-acting beta agonist and inhaled corticosteroid responsiveness in randomised controlled trials of individuals of African descent with asthma. Lancet Child Adolesc Health. 2021 12; 5(12):862-872.
Step-Up Therapy in Black Children and Adults with Poorly Controlled Asthma. N Engl J Med. 2019 09 26; 381(13):1227-1239.
Loss of bronchoprotection with ICS plus LABA treatment, ?-receptor dynamics, and the effect of alendronate. J Allergy Clin Immunol. 2019 08; 144(2):416-425.e7.
?2-agonists and acute respiratory distress syndrome. Am J Respir Crit Care Med. 2014 Mar 15; 189(6):624-5.
Thymic stromal lymphopoietin gene promoter polymorphisms are associated with susceptibility to bronchial asthma. Am J Respir Cell Mol Biol. 2011 Jun; 44(6):787-93.
Phosphodiesterase 4 inhibition of beta2-integrin adhesion caused by leukotriene B4 and TNF-alpha in human neutrophils. Eur Respir J. 2006 Nov; 28(5):920-8.
beta(2)-Agonists for treatment of pulmonary edema: ready for clinical studies? Crit Care Med. 2004 Jul; 32(7):1607-8.
Additive blockade of beta 2-integrin adhesion of eosinophils by salmeterol and fluticasone propionate. Eur Respir J. 2004 Apr; 23(4):511-7.
Beta2-integrin adhesion caused by eotaxin but not IL-5 is blocked by PDE-4 inhibition and beta2-adrenoceptor activation in human eosinophils. Pulm Pharmacol Ther. 2004; 17(2):73-9.
Blockade of LTC4 synthesis caused by additive inhibition of gIV-PLA2 phosphorylation: Effect of salmeterol and PDE4 inhibition in human eosinophils. J Allergy Clin Immunol. 2003 Aug; 112(2):404-10.