"Receptors, Cholinergic" is a descriptor in the National Library of Medicine's controlled vocabulary thesaurus,
MeSH (Medical Subject Headings). Descriptors are arranged in a hierarchical structure,
which enables searching at various levels of specificity.
Cell surface proteins that bind acetylcholine with high affinity and trigger intracellular changes influencing the behavior of cells. Cholinergic receptors are divided into two major classes, muscarinic and nicotinic, based originally on their affinity for nicotine and muscarine. Each group is further subdivided based on pharmacology, location, mode of action, and/or molecular biology.
| Descriptor ID |
D011950
|
| MeSH Number(s) |
D12.776.543.750.720.360
|
| Concept/Terms |
Receptors, Cholinergic- Receptors, Cholinergic
- Receptors, Acetylcholine
- Cholinoceptive Sites
- Sites, Cholinoceptive
- Receptors, ACh
- Cholinergic Receptors
- ACh Receptors
- Acetylcholine Receptors
- Cholinoceptors
|
Below are MeSH descriptors whose meaning is more general than "Receptors, Cholinergic".
Below are MeSH descriptors whose meaning is more specific than "Receptors, Cholinergic".
This graph shows the total number of publications written about "Receptors, Cholinergic" by people in this website by year, and whether "Receptors, Cholinergic" was a major or minor topic of these publications.
To see the data from this visualization as text,
click here.
| Year | Major Topic | Minor Topic | Total |
|---|
| 1994 | 2 | 0 | 2 |
| 1995 | 1 | 0 | 1 |
| 1996 | 2 | 0 | 2 |
| 1997 | 2 | 1 | 3 |
| 1998 | 2 | 1 | 3 |
| 1999 | 0 | 2 | 2 |
| 2002 | 0 | 1 | 1 |
| 2003 | 1 | 1 | 2 |
| 2004 | 1 | 1 | 2 |
| 2006 | 2 | 1 | 3 |
| 2007 | 1 | 0 | 1 |
| 2008 | 1 | 0 | 1 |
| 2010 | 1 | 0 | 1 |
| 2011 | 1 | 3 | 4 |
| 2014 | 1 | 1 | 2 |
| 2016 | 0 | 1 | 1 |
| 2024 | 1 | 0 | 1 |
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Below are the most recent publications written about "Receptors, Cholinergic" by people in Profiles.
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The severity of MUSK pathogenic variants is predicted by the protein domain they disrupt. HGG Adv. 2024 Jul 18; 5(3):100288.
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Impaired regulatory B cells in myasthenia gravis. J Neuroimmunol. 2016 08 15; 297:38-45.
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Optogenetic measurement of presynaptic calcium transients using conditional genetically encoded calcium indicator expression in dopaminergic neurons. PLoS One. 2014; 9(10):e111749.
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CD1d(hi)CD5+ B cells expanded by GM-CSF in vivo suppress experimental autoimmune myasthenia gravis. J Immunol. 2014 Sep 15; 193(6):2669-77.
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Defining modulatory inputs into CNS neuronal subclasses by functional pharmacological profiling. Proc Natl Acad Sci U S A. 2014 Apr 29; 111(17):6449-54.
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LG2 agrin mutation causing severe congenital myasthenic syndrome mimics functional characteristics of non-neural (z-) agrin. Hum Genet. 2012 Jul; 131(7):1123-35.
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Acute severe animal model of anti-muscle-specific kinase myasthenia: combined postsynaptic and presynaptic changes. Arch Neurol. 2012 Apr; 69(4):453-60.
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Coordinated regulation of cholinergic motor neuron traits through a conserved terminal selector gene. Nat Neurosci. 2011 Nov 27; 15(2):205-14.
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Skeletal muscle IP3R1 receptors amplify physiological and pathological synaptic calcium signals. J Neurosci. 2011 Oct 26; 31(43):15269-83.
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Mutations in MUSK causing congenital myasthenic syndrome impair MuSK-Dok-7 interaction. Hum Mol Genet. 2010 Jun 15; 19(12):2370-9.