"Calcium Channels, N-Type" is a descriptor in the National Library of Medicine's controlled vocabulary thesaurus,
MeSH (Medical Subject Headings). Descriptors are arranged in a hierarchical structure,
which enables searching at various levels of specificity.
CALCIUM CHANNELS that are concentrated in neural tissue. Omega toxins inhibit the actions of these channels by altering their voltage dependence.
| Descriptor ID |
D020864
|
| MeSH Number(s) |
D12.776.157.530.400.150.585 D12.776.543.550.450.150.585 D12.776.543.585.400.150.585
|
| Concept/Terms |
Calcium Channels, N-Type- Calcium Channels, N-Type
- Calcium Channels, N Type
- Channels, N-Type Calcium
- N-Type VDCC
- N Type VDCC
- VDCC, N-Type
- N-Type Calcium Channel
- Calcium Channel, N-Type
- Channel, N-Type Calcium
- N Type Calcium Channel
- N-Type Voltage-Dependent Calcium Channels
- N Type Voltage Dependent Calcium Channels
- N-Type Calcium Channels
- N Type Calcium Channels
- Neural-Type Calcium Channels
- Calcium Channels, Neural-Type
- Channels, Neural-Type Calcium
- Neural Type Calcium Channels
|
Below are MeSH descriptors whose meaning is more general than "Calcium Channels, N-Type".
Below are MeSH descriptors whose meaning is more specific than "Calcium Channels, N-Type".
This graph shows the total number of publications written about "Calcium Channels, N-Type" by people in this website by year, and whether "Calcium Channels, N-Type" was a major or minor topic of these publications.
To see the data from this visualization as text,
click here.
| Year | Major Topic | Minor Topic | Total |
|---|
| 1997 | 0 | 1 | 1 |
| 2000 | 0 | 1 | 1 |
| 2001 | 1 | 2 | 3 |
| 2002 | 1 | 0 | 1 |
| 2003 | 0 | 2 | 2 |
| 2005 | 0 | 1 | 1 |
| 2006 | 1 | 1 | 2 |
| 2007 | 1 | 1 | 2 |
| 2008 | 1 | 1 | 2 |
| 2010 | 0 | 1 | 1 |
| 2012 | 0 | 1 | 1 |
| 2013 | 1 | 1 | 2 |
| 2015 | 0 | 1 | 1 |
| 2016 | 0 | 2 | 2 |
| 2018 | 0 | 2 | 2 |
| 2019 | 1 | 0 | 1 |
To return to the timeline,
click here.
Below are the most recent publications written about "Calcium Channels, N-Type" by people in Profiles.
-
a1ACT Is Essential for Survival and Early Cerebellar Programming in a Critical Neonatal Window. Neuron. 2019 05 22; 102(4):770-785.e7.
-
Targeting the CACNA1A IRES as a Treatment for Spinocerebellar Ataxia Type 6. Cerebellum. 2018 02; 17(1):72-77.
-
Spinocerebellar [corrected] Ataxia Type 6: Molecular Mechanisms and Calcium Channel Genetics. Adv Exp Med Biol. 2018; 1049:147-173.
-
Evaluation of parkinsonism and striatal dopamine transporter loss in patients with spinocerebellar ataxia type 6. J Neurol. 2016 Nov; 263(11):2302-2307.
-
An miRNA-mediated therapy for SCA6 blocks IRES-driven translation of the CACNA1A second cistron. Sci Transl Med. 2016 07 13; 8(347):347ra94.
-
DnaJ-1 and karyopherin a3 suppress degeneration in a new Drosophila model of Spinocerebellar Ataxia Type 6. Hum Mol Genet. 2015 Aug 01; 24(15):4385-96.
-
Second cistron in CACNA1A gene encodes a transcription factor mediating cerebellar development and SCA6. Cell. 2013 Jul 03; 154(1):118-33.
-
Stable respiratory activity requires both P/Q-type and N-type voltage-gated calcium channels. J Neurosci. 2013 Feb 20; 33(8):3633-45.
-
Spinocerebellar ataxia type 6. Handb Clin Neurol. 2012; 103:461-73.
-
Presynaptic activity and CaMKII modulate retrograde semaphorin signaling and synaptic refinement. Neuron. 2010 Oct 06; 68(1):32-44.