Sister Chromatid Exchange
"Sister Chromatid Exchange" is a descriptor in the National Library of Medicine's controlled vocabulary thesaurus,
MeSH (Medical Subject Headings). Descriptors are arranged in a hierarchical structure,
which enables searching at various levels of specificity.
An exchange of segments between the sister chromatids of a chromosome, either between the sister chromatids of a meiotic tetrad or between the sister chromatids of a duplicated somatic chromosome. Its frequency is increased by ultraviolet and ionizing radiation and other mutagenic agents and is particularly high in BLOOM SYNDROME.
| Descriptor ID |
D012854
|
| MeSH Number(s) |
G05.728.615.750
|
| Concept/Terms |
Sister Chromatid Exchange- Sister Chromatid Exchange
- Chromatid Exchange, Sister
- Chromatid Exchanges, Sister
- Exchange, Sister Chromatid
- Exchanges, Sister Chromatid
- Sister Chromatid Exchanges
|
Below are MeSH descriptors whose meaning is more general than "Sister Chromatid Exchange".
Below are MeSH descriptors whose meaning is more specific than "Sister Chromatid Exchange".
This graph shows the total number of publications written about "Sister Chromatid Exchange" by people in this website by year, and whether "Sister Chromatid Exchange" was a major or minor topic of these publications.
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| Year | Major Topic | Minor Topic | Total |
|---|
| 2011 | 1 | 0 | 1 |
| 2015 | 0 | 1 | 1 |
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Below are the most recent publications written about "Sister Chromatid Exchange" by people in Profiles.
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Small Rad51 and Dmc1 Complexes Often Co-occupy Both Ends of a Meiotic DNA Double Strand Break. PLoS Genet. 2015 Dec; 11(12):e1005653.
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Trex2 enables spontaneous sister chromatid exchanges without facilitating DNA double-strand break repair. Genetics. 2011 Aug; 188(4):787-97.
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Human Mus81 and FANCB independently contribute to repair of DNA damage during replication. Genes Cells. 2007 Oct; 12(10):1111-22.
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Mice lacking sister chromatid cohesion protein PDS5B exhibit developmental abnormalities reminiscent of Cornelia de Lange syndrome. Development. 2007 Sep; 134(17):3191-201.
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Absence of p53 enhances growth defects and etoposide sensitivity of human cells lacking the Bloom syndrome helicase BLM. DNA Cell Biol. 2007 Jul; 26(7):517-25.
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Fen-1 facilitates homologous recombination by removing divergent sequences at DNA break ends. Mol Cell Biol. 2005 Aug; 25(16):6948-55.
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Genetic interactions between BLM and DNA ligase IV in human cells. J Biol Chem. 2004 Dec 31; 279(53):55433-42.
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Enhanced tumor formation in mice heterozygous for Blm mutation. Science. 2002 Sep 20; 297(5589):2051-3.
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Chromosomal sensitivity of lymphocytes from individuals with therapy-related acute nonlymphocytic leukemia. Mutat Res. 1989 Apr; 216(2):119-26.
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Cytogenetic observations of a human ovarian carcinoma clinically resistant to therapy. Am J Obstet Gynecol. 1988 Nov; 159(5):1099-103.