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Connection

Nanduri R. Prabhakar to Rats, Sprague-Dawley

This is a "connection" page, showing publications Nanduri R. Prabhakar has written about Rats, Sprague-Dawley.
Connection Strength

2.943
  1. Long-term facilitation of catecholamine secretion from adrenal chromaffin cells of neonatal rats by chronic intermittent hypoxia. J Neurophysiol. 2019 11 01; 122(5):1874-1883.
    View in: PubMed
    Score: 0.136
  2. H2S mediates carotid body response to hypoxia but not anoxia. Respir Physiol Neurobiol. 2019 01; 259:75-85.
    View in: PubMed
    Score: 0.126
  3. DNA methylation in the central and efferent limbs of the chemoreflex requires carotid body neural activity. J Physiol. 2018 08; 596(15):3087-3100.
    View in: PubMed
    Score: 0.121
  4. Epigenetic regulation of redox state mediates persistent cardiorespiratory abnormalities after long-term intermittent hypoxia. J Physiol. 2017 01 01; 595(1):63-77.
    View in: PubMed
    Score: 0.111
  5. CaV3.2 T-type Ca2+ channels mediate the augmented calcium influx in carotid body glomus cells by chronic intermittent hypoxia. J Neurophysiol. 2016 Jan 01; 115(1):345-54.
    View in: PubMed
    Score: 0.104
  6. Carotid Body Chemoreflex Mediates Intermittent Hypoxia-Induced Oxidative Stress in the Adrenal Medulla. Adv Exp Med Biol. 2015; 860:195-9.
    View in: PubMed
    Score: 0.098
  7. CaV3.2 T-type Ca²? channels in H2S-mediated hypoxic response of the carotid body. Am J Physiol Cell Physiol. 2015 Jan 15; 308(2):C146-54.
    View in: PubMed
    Score: 0.097
  8. Regulation of hypoxia-inducible factor-a isoforms and redox state by carotid body neural activity in rats. J Physiol. 2014 Sep 01; 592(17):3841-58.
    View in: PubMed
    Score: 0.095
  9. Hypoxia-inducible factors regulate human and rat cystathionine ß-synthase gene expression. Biochem J. 2014 Mar 01; 458(2):203-11.
    View in: PubMed
    Score: 0.093
  10. Inherent variations in CO-H2S-mediated carotid body O2 sensing mediate hypertension and pulmonary edema. Proc Natl Acad Sci U S A. 2014 Jan 21; 111(3):1174-9.
    View in: PubMed
    Score: 0.092
  11. Xanthine oxidase mediates hypoxia-inducible factor-2a degradation by intermittent hypoxia. PLoS One. 2013; 8(10):e75838.
    View in: PubMed
    Score: 0.090
  12. Role of oxidative stress-induced endothelin-converting enzyme activity in the alteration of carotid body function by chronic intermittent hypoxia. Exp Physiol. 2013 Nov; 98(11):1620-30.
    View in: PubMed
    Score: 0.089
  13. Endogenous H2S is required for hypoxic sensing by carotid body glomus cells. Am J Physiol Cell Physiol. 2012 Nov 01; 303(9):C916-23.
    View in: PubMed
    Score: 0.083
  14. Endothelin-1 mediates attenuated carotid baroreceptor activity by intermittent hypoxia. J Appl Physiol (1985). 2012 Jan; 112(1):187-96.
    View in: PubMed
    Score: 0.079
  15. Angiotensin II evokes sensory long-term facilitation of the carotid body via NADPH oxidase. J Appl Physiol (1985). 2011 Oct; 111(4):964-70.
    View in: PubMed
    Score: 0.077
  16. NADPH oxidase 2 mediates intermittent hypoxia-induced mitochondrial complex I inhibition: relevance to blood pressure changes in rats. Antioxid Redox Signal. 2011 Feb 15; 14(4):533-42.
    View in: PubMed
    Score: 0.073
  17. Neonatal intermittent hypoxia impairs neuronal nicotinic receptor expression and function in adrenal chromaffin cells. Am J Physiol Cell Physiol. 2010 Aug; 299(2):C381-8.
    View in: PubMed
    Score: 0.072
  18. NADPH oxidase is required for the sensory plasticity of the carotid body by chronic intermittent hypoxia. J Neurosci. 2009 Apr 15; 29(15):4903-10.
    View in: PubMed
    Score: 0.066
  19. Neonatal intermittent hypoxia leads to long-lasting facilitation of acute hypoxia-evoked catecholamine secretion from rat chromaffin cells. J Neurophysiol. 2009 Jun; 101(6):2837-46.
    View in: PubMed
    Score: 0.066
  20. Intermittent hypoxia degrades HIF-2alpha via calpains resulting in oxidative stress: implications for recurrent apnea-induced morbidities. Proc Natl Acad Sci U S A. 2009 Jan 27; 106(4):1199-204.
    View in: PubMed
    Score: 0.065
  21. Contrasting effects of intermittent and continuous hypoxia on low O(2) evoked catecholamine secretion from neonatal rat chromaffin cells. Adv Exp Med Biol. 2009; 648:345-9.
    View in: PubMed
    Score: 0.065
  22. Reactive oxygen species-dependent endothelin signaling is required for augmented hypoxic sensory response of the neonatal carotid body by intermittent hypoxia. Am J Physiol Regul Integr Comp Physiol. 2009 Mar; 296(3):R735-42.
    View in: PubMed
    Score: 0.065
  23. Comparative analysis of neonatal and adult rat carotid body responses to chronic intermittent hypoxia. J Appl Physiol (1985). 2008 May; 104(5):1287-94.
    View in: PubMed
    Score: 0.061
  24. Acute intermittent hypoxia increases both phrenic and sympathetic nerve activities in the rat. Exp Physiol. 2007 Jan; 92(1):87-97.
    View in: PubMed
    Score: 0.056
  25. 5-HT evokes sensory long-term facilitation of rodent carotid body via activation of NADPH oxidase. J Physiol. 2006 Oct 01; 576(Pt 1):289-95.
    View in: PubMed
    Score: 0.055
  26. Acute lung injury augments hypoxic ventilatory response in the absence of systemic hypoxemia. J Appl Physiol (1985). 2006 Dec; 101(6):1795-802.
    View in: PubMed
    Score: 0.055
  27. Chronic intermittent hypoxia induces hypoxia-evoked catecholamine efflux in adult rat adrenal medulla via oxidative stress. J Physiol. 2006 Aug 15; 575(Pt 1):229-39.
    View in: PubMed
    Score: 0.054
  28. Modulation of the hypoxic sensory response of the carotid body by 5-hydroxytryptamine: role of the 5-HT2 receptor. Respir Physiol Neurobiol. 2005 Feb 15; 145(2-3):135-42.
    View in: PubMed
    Score: 0.050
  29. Entrainment pattern between sympathetic and phrenic nerve activities in the Sprague-Dawley rat: hypoxia-evoked sympathetic activity during expiration. Am J Physiol Regul Integr Comp Physiol. 2004 Jun; 286(6):R1121-8.
    View in: PubMed
    Score: 0.046
  30. Transcriptomic Analysis of Postnatal Rat Carotid Body Development. Genes (Basel). 2024 02 27; 15(3).
    View in: PubMed
    Score: 0.046
  31. Detection of oxygen sensing during intermittent hypoxia. Methods Enzymol. 2004; 381:107-20.
    View in: PubMed
    Score: 0.046
  32. Effect of two paradigms of chronic intermittent hypoxia on carotid body sensory activity. J Appl Physiol (1985). 2004 Mar; 96(3):1236-42; discussion 1196.
    View in: PubMed
    Score: 0.046
  33. Induction of sensory long-term facilitation in the carotid body by intermittent hypoxia: implications for recurrent apneas. Proc Natl Acad Sci U S A. 2003 Aug 19; 100(17):10073-8.
    View in: PubMed
    Score: 0.045
  34. Reactive oxygen species in the plasticity of respiratory behavior elicited by chronic intermittent hypoxia. J Appl Physiol (1985). 2003 Jun; 94(6):2342-9.
    View in: PubMed
    Score: 0.043
  35. Systemic and cellular responses to intermittent hypoxia: evidence for oxidative stress and mitochondrial dysfunction. Adv Exp Med Biol. 2003; 536:559-64.
    View in: PubMed
    Score: 0.043
  36. Selected Contribution: Improved anoxic tolerance in rat diaphragm following intermittent hypoxia. J Appl Physiol (1985). 2001 Jun; 90(6):2508-13.
    View in: PubMed
    Score: 0.038
  37. Chronic intermittent hypoxia enhances carotid body chemoreceptor response to low oxygen. Adv Exp Med Biol. 2001; 499:33-8.
    View in: PubMed
    Score: 0.037
  38. Activation of nitric oxide synthase gene expression by hypoxia in central and peripheral neurons. Brain Res Mol Brain Res. 1996 Dec 31; 43(1-2):341-6.
    View in: PubMed
    Score: 0.028
  39. Heterogeneity in cytosolic calcium responses to hypoxia in carotid body cells. Brain Res. 1996 Jan 15; 706(2):297-302.
    View in: PubMed
    Score: 0.026
  40. Regulation of neuronal nitric oxide synthase gene expression by hypoxia. Role of nitric oxide in respiratory adaptation to low pO2. Adv Exp Med Biol. 1996; 410:345-8.
    View in: PubMed
    Score: 0.026
  41. Calpain activation by ROS mediates human ether-a-go-go-related gene protein degradation by intermittent hypoxia. Am J Physiol Cell Physiol. 2016 Mar 01; 310(5):C329-36.
    View in: PubMed
    Score: 0.026
  42. Carbon monoxide: a role in carotid body chemoreception. Proc Natl Acad Sci U S A. 1995 Mar 14; 92(6):1994-7.
    View in: PubMed
    Score: 0.025
  43. Tachykinin antagonists in carotid body responses to hypoxia and substance P in the rat. Respir Physiol. 1994 Mar; 95(3):295-310.
    View in: PubMed
    Score: 0.023
  44. Role of substance P in rat carotid body responses to hypoxia and capsaicin. Adv Exp Med Biol. 1993; 337:265-70.
    View in: PubMed
    Score: 0.021
  45. NADPH oxidase-derived H(2)O(2) contributes to angiotensin II-induced aldosterone synthesis in human and rat adrenal cortical cells. Antioxid Redox Signal. 2012 Aug 01; 17(3):445-59.
    View in: PubMed
    Score: 0.020
  46. Enhanced neuropeptide Y synthesis during intermittent hypoxia in the rat adrenal medulla: role of reactive oxygen species-dependent alterations in precursor peptide processing. Antioxid Redox Signal. 2011 Apr 01; 14(7):1179-90.
    View in: PubMed
    Score: 0.019
  47. Pattern-specific sustained activation of tyrosine hydroxylase by intermittent hypoxia: role of reactive oxygen species-dependent downregulation of protein phosphatase 2A and upregulation of protein kinases. Antioxid Redox Signal. 2009 Aug; 11(8):1777-89.
    View in: PubMed
    Score: 0.017
  48. Intermittent hypoxia activates peptidylglycine alpha-amidating monooxygenase in rat brain stem via reactive oxygen species-mediated proteolytic processing. J Appl Physiol (1985). 2009 Jan; 106(1):12-9.
    View in: PubMed
    Score: 0.016
  49. Nitric oxide and ventilatory response to hypoxia. Respir Physiol. 1995 Sep; 101(3):257-66.
    View in: PubMed
    Score: 0.006
  50. Low PO2 dependency of neutral endopeptidase and acetylcholinesterase activities of the rat carotid body. Adv Exp Med Biol. 1994; 360:217-20.
    View in: PubMed
    Score: 0.006
Connection Strength

The connection strength for concepts is the sum of the scores for each matching publication.

Publication scores are based on many factors, including how long ago they were written and whether the person is a first or senior author.