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Mice, Mutant StrainsConcept Why?
Alterations in dopamine release but not dopamine autoreceptor function in dopamine D3 receptor mutant mice.Academic Article Why?
Growth requirements of B lineage lymphocytes from scid and normal mice.Academic Article Why?
Mouse forward genetics in the study of the peripheral nervous system and human peripheral neuropathy.Academic Article Why?
Nephron-deficient Fvb mice develop rapidly progressive renal failure and heavy albuminuria involving excess glomerular GLUT1 and VEGF.Academic Article Why?
Sequences of immunoglobulin lambda 1 genes in a lambda 1 defective mouse strain.Academic Article Why?
The role of autoreactive T-cell lines in the restoration of the immune response to sheep red blood cells in C57Bl/6-lpr/lpr mice.Academic Article Why?
Xu, MingPerson Why?
Beyer, Eric C.Person Why?
?40 Isoform of p53 controls ß-cell proliferation and glucose homeostasis in mice.Academic Article Why?
Alpha 2-adrenergic receptor subtypes: subtle mutation of the alpha 2A-adrenergic receptor in vivo by gene targeting strategies reveals the role of this subtype in multiple physiological settings.Academic Article Why?
Biochemical correlations in animal models of chondrodysplasias.Academic Article Why?
Calcium threshold shift enables frequency-independent control of plasticity by an instructive signal.Academic Article Why?
CD95 signaling deficient mice with a wild-type hematopoietic system are prone to hepatic neoplasia.Academic Article Why?
Cellular responses to psychomotor stimulant and neuroleptic drugs are abnormal in mice lacking the D1 dopamine receptor.Academic Article Why?
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