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The degradation of promyelocytic leukemia and Sp100 proteins by herpes simplex virus 1 is mediated by the ubiquitin-conjugating enzyme UbcH5a.
PML plays both inimical and beneficial roles in HSV-1 replication.
The SP100 component of ND10 enhances accumulation of PML and suppresses replication and the assembly of HSV replication compartments.