"Mice, Inbred C57BL" is a descriptor in the National Library of Medicine's controlled vocabulary thesaurus,
MeSH (Medical Subject Headings). Descriptors are arranged in a hierarchical structure,
which enables searching at various levels of specificity.
One of the first INBRED MOUSE STRAINS to be sequenced. This strain is commonly used as genetic background for transgenic mouse models. Refractory to many tumors, this strain is also preferred model for studying role of genetic variations in development of diseases.
Descriptor ID |
D008810
|
MeSH Number(s) |
B01.050.050.199.520.520.420 B01.050.150.900.649.313.992.635.505.500.400.420
|
Concept/Terms |
Mice, Inbred C57BL- Mice, Inbred C57BL
- C57BL Mice, Inbred
- Inbred C57BL Mice
- Mouse, Inbred C57BL
- C57BL Mouse, Inbred
- Inbred C57BL Mouse
- Mice, C57BL
- C57BL Mice
- Mouse, C57BL
- C57BL Mouse
|
Below are MeSH descriptors whose meaning is more general than "Mice, Inbred C57BL".
Below are MeSH descriptors whose meaning is more specific than "Mice, Inbred C57BL".
This graph shows the total number of publications written about "Mice, Inbred C57BL" by people in this website by year, and whether "Mice, Inbred C57BL" was a major or minor topic of these publications.
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Year | Major Topic | Minor Topic | Total |
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1994 | 0 | 17 | 17 |
1995 | 0 | 28 | 28 |
1996 | 0 | 18 | 18 |
1997 | 0 | 29 | 29 |
1998 | 1 | 36 | 37 |
1999 | 0 | 35 | 35 |
2000 | 0 | 31 | 31 |
2001 | 1 | 48 | 49 |
2002 | 0 | 53 | 53 |
2003 | 0 | 64 | 64 |
2004 | 0 | 58 | 58 |
2005 | 1 | 71 | 72 |
2006 | 1 | 71 | 72 |
2007 | 1 | 71 | 72 |
2008 | 0 | 71 | 71 |
2009 | 2 | 97 | 99 |
2010 | 1 | 116 | 117 |
2011 | 0 | 129 | 129 |
2012 | 0 | 130 | 130 |
2013 | 0 | 124 | 124 |
2014 | 0 | 127 | 127 |
2015 | 0 | 132 | 132 |
2016 | 0 | 108 | 108 |
2017 | 0 | 103 | 103 |
2018 | 0 | 85 | 85 |
2019 | 0 | 93 | 93 |
2020 | 0 | 102 | 102 |
2021 | 0 | 79 | 79 |
2022 | 0 | 22 | 22 |
2023 | 0 | 17 | 17 |
2024 | 5 | 25 | 30 |
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Below are the most recent publications written about "Mice, Inbred C57BL" by people in Profiles.
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High-throughput screen identifies non inflammatory small molecule inducers of trained immunity. Proc Natl Acad Sci U S A. 2024 Jul 16; 121(29):e2400413121.
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Local, Quantitative Morphometry of Fibroproliferative Lung Injury Using Laminin. Am J Respir Cell Mol Biol. 2024 Jul; 71(1):23-29.
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Upregulated selenoprotein I during lipopolysaccharide-induced B cell activation promotes lipidomic changes and is required for effective differentiation into IgM-secreting plasma B cells. J Leukoc Biol. 2024 Jun 28; 116(1):6-17.
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PIKfyve, expressed by CD11c-positive cells, controls tumor immunity. Nat Commun. 2024 Jun 28; 15(1):5487.
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Eomes expression identifies the early bone marrow precursor to classical NK cells. Nat Immunol. 2024 Jul; 25(7):1172-1182.
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Deficiency in non-classical major histocompatibility class II-like molecule, H2-O confers protection against Staphylococcus aureus in mice. PLoS Pathog. 2024 Jun; 20(6):e1012306.
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Intrinsic and synaptic determinants of receptive field plasticity in Purkinje cells of the mouse cerebellum. Nat Commun. 2024 May 31; 15(1):4645.
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Pervasive nuclear envelope ruptures precede ECM signaling and disease onset without activating cGAS-STING in Lamin-cardiomyopathy mice. Cell Rep. 2024 Jun 25; 43(6):114284.
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Genetic Background Strongly Influences the Impact of Carrying the Thr92Ala-DIO2 Polymorphism in the Male Mouse. Endocrinology. 2024 May 27; 165(7).
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Interaction between the gut microbiota and colonic enteroendocrine cells regulates host metabolism. Nat Metab. 2024 Jun; 6(6):1076-1091.