"Mice, Inbred NOD" is a descriptor in the National Library of Medicine's controlled vocabulary thesaurus,
MeSH (Medical Subject Headings). Descriptors are arranged in a hierarchical structure,
which enables searching at various levels of specificity.
A strain of non-obese diabetic mice developed in Japan that has been widely studied as a model for T-cell-dependent autoimmune insulin-dependent diabetes mellitus in which insulitis is a major histopathologic feature, and in which genetic susceptibility is strongly MHC-linked.
| Descriptor ID |
D016688
|
| MeSH Number(s) |
B01.050.050.199.520.520.565 B01.050.150.900.649.313.992.635.505.500.400.565
|
| Concept/Terms |
Mice, Inbred NOD- Mice, Inbred NOD
- Inbred NOD Mice
- NOD Mice, Inbred
- Mouse, NOD
- NOD Mouse
- Nonobese Diabetic Mice
- Diabetic Mice, Nonobese
- Mice, Nonobese Diabetic
- Non-Obese Diabetic Mouse
- Diabetic Mouse, Non-Obese
- Mouse, Non-Obese Diabetic
- Non Obese Diabetic Mouse
- Non-Obese Diabetic Mice
- Diabetic Mice, Non-Obese
- Mice, Non-Obese Diabetic
- Non Obese Diabetic Mice
- Mouse, Inbred NOD
- Inbred NOD Mouse
- NOD Mouse, Inbred
- Mice, NOD
- NOD Mice
- Nonobese Diabetic Mouse
- Diabetic Mouse, Nonobese
- Mouse, Nonobese Diabetic
|
Below are MeSH descriptors whose meaning is more general than "Mice, Inbred NOD".
Below are MeSH descriptors whose meaning is more specific than "Mice, Inbred NOD".
This graph shows the total number of publications written about "Mice, Inbred NOD" by people in this website by year, and whether "Mice, Inbred NOD" was a major or minor topic of these publications.
To see the data from this visualization as text,
click here.
| Year | Major Topic | Minor Topic | Total |
|---|
| 1995 | 0 | 1 | 1 |
| 1996 | 0 | 1 | 1 |
| 1997 | 0 | 3 | 3 |
| 1998 | 1 | 1 | 2 |
| 1999 | 0 | 3 | 3 |
| 2000 | 0 | 1 | 1 |
| 2001 | 1 | 4 | 5 |
| 2002 | 0 | 2 | 2 |
| 2003 | 0 | 3 | 3 |
| 2004 | 0 | 4 | 4 |
| 2005 | 0 | 5 | 5 |
| 2006 | 0 | 4 | 4 |
| 2007 | 0 | 3 | 3 |
| 2008 | 0 | 6 | 6 |
| 2009 | 0 | 5 | 5 |
| 2010 | 0 | 7 | 7 |
| 2011 | 0 | 2 | 2 |
| 2012 | 0 | 7 | 7 |
| 2013 | 0 | 10 | 10 |
| 2014 | 0 | 8 | 8 |
| 2015 | 0 | 8 | 8 |
| 2016 | 0 | 11 | 11 |
| 2017 | 0 | 3 | 3 |
| 2018 | 0 | 12 | 12 |
| 2019 | 0 | 19 | 19 |
| 2020 | 0 | 8 | 8 |
| 2021 | 0 | 2 | 2 |
| 2022 | 0 | 3 | 3 |
| 2023 | 0 | 1 | 1 |
| 2024 | 0 | 3 | 3 |
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Below are the most recent publications written about "Mice, Inbred NOD" by people in Profiles.
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Androgens contribute to sex bias of autoimmunity in mice by T cell-intrinsic regulation of Ptpn22 phosphatase expression. Nat Commun. 2024 Sep 03; 15(1):7688.
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Inhibition of the eukaryotic initiation factor-2a kinase PERK decreases risk of autoimmune diabetes in mice. J Clin Invest. 2024 Jun 18; 134(16).
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Tissue-infiltrating alloreactive T cells require Id3 to deflect PD-1-mediated immune suppression during GVHD. Blood. 2024 01 11; 143(2):166-177.
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Microbiota-dependent proteolysis of gluten subverts diet-mediated protection against type 1 diabetes. Cell Host Microbe. 2023 02 08; 31(2):213-227.e9.
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Co-clinical Modeling of the Activity of the BET Inhibitor Mivebresib (ABBV-075) in AML. In Vivo. 2022 Jul-Aug; 36(4):1615-1627.
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Proinflammatory signaling in islet ß cells propagates invasion of pathogenic immune cells in autoimmune diabetes. Cell Rep. 2022 06 28; 39(13):111011.
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Positive and negative selection shape the human naive B cell repertoire. J Clin Invest. 2022 01 18; 132(2).
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HRD1-mediated METTL14 degradation regulates m6A mRNA modification to suppress ER proteotoxic liver disease. Mol Cell. 2021 12 16; 81(24):5052-5065.e6.
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Lysine acetylation restricts mutant IDH2 activity to optimize transformation in AML cells. Mol Cell. 2021 09 16; 81(18):3833-3847.e11.
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Concurrent Targeting of Potential Cancer Stem Cells Regulating Pathways Sensitizes Lung Adenocarcinoma to Standard Chemotherapy. Mol Cancer Ther. 2020 10; 19(10):2175-2185.