"Mice, Inbred NOD" is a descriptor in the National Library of Medicine's controlled vocabulary thesaurus,
MeSH (Medical Subject Headings). Descriptors are arranged in a hierarchical structure,
which enables searching at various levels of specificity.
A strain of non-obese diabetic mice developed in Japan that has been widely studied as a model for T-cell-dependent autoimmune insulin-dependent diabetes mellitus in which insulitis is a major histopathologic feature, and in which genetic susceptibility is strongly MHC-linked.
| Descriptor ID |
D016688
|
| MeSH Number(s) |
B01.050.050.199.520.520.565 B01.050.150.900.649.313.992.635.505.500.400.565
|
| Concept/Terms |
Mice, Inbred NOD- Mice, Inbred NOD
- Inbred NOD Mice
- NOD Mice, Inbred
- Mouse, NOD
- NOD Mouse
- Nonobese Diabetic Mice
- Diabetic Mice, Nonobese
- Mice, Nonobese Diabetic
- Non-Obese Diabetic Mouse
- Diabetic Mouse, Non-Obese
- Mouse, Non-Obese Diabetic
- Non Obese Diabetic Mouse
- Non-Obese Diabetic Mice
- Diabetic Mice, Non-Obese
- Mice, Non-Obese Diabetic
- Non Obese Diabetic Mice
- Mouse, Inbred NOD
- Inbred NOD Mouse
- NOD Mouse, Inbred
- Mice, NOD
- NOD Mice
- Nonobese Diabetic Mouse
- Diabetic Mouse, Nonobese
- Mouse, Nonobese Diabetic
|
Below are MeSH descriptors whose meaning is more general than "Mice, Inbred NOD".
Below are MeSH descriptors whose meaning is more specific than "Mice, Inbred NOD".
This graph shows the total number of publications written about "Mice, Inbred NOD" by people in this website by year, and whether "Mice, Inbred NOD" was a major or minor topic of these publications.
To see the data from this visualization as text,
click here.
| Year | Major Topic | Minor Topic | Total |
|---|
| 1996 | 0 | 1 | 1 |
| 1997 | 0 | 5 | 5 |
| 1998 | 1 | 1 | 2 |
| 1999 | 0 | 3 | 3 |
| 2000 | 0 | 1 | 1 |
| 2001 | 1 | 4 | 5 |
| 2002 | 0 | 2 | 2 |
| 2003 | 0 | 3 | 3 |
| 2004 | 0 | 4 | 4 |
| 2005 | 0 | 5 | 5 |
| 2006 | 0 | 4 | 4 |
| 2007 | 0 | 3 | 3 |
| 2008 | 0 | 6 | 6 |
| 2009 | 0 | 5 | 5 |
| 2010 | 0 | 8 | 8 |
| 2011 | 0 | 2 | 2 |
| 2012 | 0 | 7 | 7 |
| 2013 | 0 | 12 | 12 |
| 2014 | 0 | 8 | 8 |
| 2015 | 0 | 10 | 10 |
| 2016 | 0 | 13 | 13 |
| 2017 | 0 | 3 | 3 |
| 2018 | 0 | 13 | 13 |
| 2019 | 0 | 21 | 21 |
| 2020 | 0 | 9 | 9 |
| 2021 | 0 | 2 | 2 |
| 2022 | 0 | 4 | 4 |
| 2023 | 0 | 1 | 1 |
| 2024 | 0 | 4 | 4 |
| 2025 | 0 | 2 | 2 |
To return to the timeline,
click here.
Below are the most recent publications written about "Mice, Inbred NOD" by people in Profiles.
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The integrated stress response promotes macrophage inflammation and migration in autoimmune diabetes. Cell Commun Signal. 2025 Aug 20; 23(1):374.
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Secretion of a VEGF-Blocking scFv Enhances CAR T-cell Potency. Cancer Immunol Res. 2025 Aug 01; 13(8):1132-1144.
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12-Lipoxygenase inhibition delays onset of autoimmune diabetes in human gene replacement mice. JCI Insight. 2024 Dec 20; 9(24).
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Androgens contribute to sex bias of autoimmunity in mice by T cell-intrinsic regulation of Ptpn22 phosphatase expression. Nat Commun. 2024 09 03; 15(1):7688.
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Inhibition of the eukaryotic initiation factor-2a kinase PERK decreases risk of autoimmune diabetes in mice. J Clin Invest. 2024 Jun 18; 134(16).
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Tissue-infiltrating alloreactive T cells require Id3 to deflect PD-1-mediated immune suppression during GVHD. Blood. 2024 01 11; 143(2):166-177.
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Microbiota-dependent proteolysis of gluten subverts diet-mediated protection against type 1 diabetes. Cell Host Microbe. 2023 02 08; 31(2):213-227.e9.
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Co-clinical Modeling of the Activity of the BET Inhibitor Mivebresib (ABBV-075) in AML. In Vivo. 2022 Jul-Aug; 36(4):1615-1627.
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Proinflammatory signaling in islet ß cells propagates invasion of pathogenic immune cells in autoimmune diabetes. Cell Rep. 2022 06 28; 39(13):111011.
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METTL16 exerts an m6A-independent function to facilitate translation and tumorigenesis. Nat Cell Biol. 2022 02; 24(2):205-216.