"Venoms" is a descriptor in the National Library of Medicine's controlled vocabulary thesaurus,
MeSH (Medical Subject Headings). Descriptors are arranged in a hierarchical structure,
which enables searching at various levels of specificity.
Poisonous animal secretions forming fluid mixtures of many different enzymes, toxins, and other substances. These substances are produced in specialized glands and secreted through specialized delivery systems (nematocysts, spines, fangs, etc.) for disabling prey or predator.
Descriptor ID |
D014688
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MeSH Number(s) |
A12.200.935 D20.888 D23.946.833
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Concept/Terms |
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Below are MeSH descriptors whose meaning is more general than "Venoms".
Below are MeSH descriptors whose meaning is more specific than "Venoms".
This graph shows the total number of publications written about "Venoms" by people in this website by year, and whether "Venoms" was a major or minor topic of these publications.
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Year | Major Topic | Minor Topic | Total |
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2005 | 1 | 0 | 1 |
2006 | 2 | 0 | 2 |
2007 | 1 | 0 | 1 |
2011 | 0 | 1 | 1 |
2013 | 0 | 1 | 1 |
2016 | 1 | 0 | 1 |
2020 | 0 | 1 | 1 |
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Below are the most recent publications written about "Venoms" by people in Profiles.
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Exenatide extended release in patients with type 1 diabetes with and without residual insulin production. Diabetes Obes Metab. 2020 11; 22(11):2045-2054.
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Glucagon-Like Peptide 1 Receptor Activation Attenuates Platelet Aggregation and Thrombosis. Diabetes. 2016 06; 65(6):1714-23.
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ß-Cell-specific protein kinase A activation enhances the efficiency of glucose control by increasing acute-phase insulin secretion. Diabetes. 2013 May; 62(5):1527-36.
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Cardiovascular effects of glucagonlike peptide-1 agonists. Am J Cardiol. 2011 Aug 02; 108(3 Suppl):33B-41B.
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Protection of pancreatic beta-cells by exendin-4 may involve the reduction of endoplasmic reticulum stress; in vivo and in vitro studies. J Endocrinol. 2007 Apr; 193(1):65-74.
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Exendin 4 controls insulin production in rat islet beta cells predominantly by potentiation of glucose-stimulated proinsulin biosynthesis at the translational level. Diabetologia. 2006 Dec; 49(12):2920-9.
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Site-3 toxins and cardiac sodium channels. Toxicon. 2007 Feb; 49(2):181-93.
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Glucagon-like peptide-1 protects beta cells from cytokine-induced apoptosis and necrosis: role of protein kinase B. Diabetologia. 2005 Jul; 48(7):1339-49.
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In vitro study on the effects of bradykinin potentiating factor and angiotensin II analogue on degradation and conversion of angiotensin I and II in plasma. Clin Chim Acta. 1974 May 17; 52(3):287-92.
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Immunosuppressive effects of anti-lymphocyte serum in complement deficient mice. II. Evidence that activity is mediated by C3 independent antibodies. J Immunol. 1974 Feb; 112(2):858-61.