"Motor Endplate" is a descriptor in the National Library of Medicine's controlled vocabulary thesaurus,
MeSH (Medical Subject Headings). Descriptors are arranged in a hierarchical structure,
which enables searching at various levels of specificity.
The specialized postsynaptic region of a muscle cell. The motor endplate is immediately across the synaptic cleft from the presynaptic axon terminal. Among its anatomical specializations are junctional folds which harbor a high density of cholinergic receptors.
- Motor Endplate
- Endplate, Motor
- Endplates, Motor
- Motor Endplates
- Motor End-Plate
- End-Plate, Motor
- End-Plates, Motor
- Motor End Plate
- Motor End-Plates
Below are MeSH descriptors whose meaning is more general than "Motor Endplate".
Below are MeSH descriptors whose meaning is more specific than "Motor Endplate".
This graph shows the total number of publications written about "Motor Endplate" by people in this website by year, and whether "Motor Endplate" was a major or minor topic of these publications.
To see the data from this visualization as text, click here.
|Year||Major Topic||Minor Topic||Total|
To return to the timeline, click here.
Below are the most recent publications written about "Motor Endplate" by people in Profiles.
A presynaptic congenital myasthenic syndrome attributed to a homozygous sequence variant in LAMA5. Ann N Y Acad Sci. 2018 02; 1413(1):119-125.
Selective inhibition of caspases in skeletal muscle reverses the apoptotic synaptic degeneration in slow-channel myasthenic syndrome. Hum Mol Genet. 2014 Jan 01; 23(1):69-77.
Synaptic basal lamina-associated congenital myasthenic syndromes. Ann N Y Acad Sci. 2012 Dec; 1275:36-48.
Focal caspase activation underlies the endplate myopathy in slow-channel syndrome. Ann Neurol. 2004 Mar; 55(3):347-52.
Monitoring neuromuscular blockade. Int Anesthesiol Clin. 2004; 42(2):25-40.
Presynaptic failure of neuromuscular transmission and synaptic remodeling in EA2. Neurology. 2003 Dec 23; 61(12):1743-8.
Active calcium accumulation underlies severe weakness in a panel of mice with slow-channel syndrome. J Neurosci. 2002 Aug 01; 22(15):6447-57.
Novel delta subunit mutation in slow-channel syndrome causes severe weakness by novel mechanisms. Ann Neurol. 2002 Jan; 51(1):102-12.
Genetic manipulation of AChR responses suggests multiple causes of weakness in slow-channel syndrome. Ann N Y Acad Sci. 1998 May 13; 841:167-80.
Desensitization of mutant acetylcholine receptors in transgenic mice reduces the amplitude of neuromuscular synaptic currents. Synapse. 1997 Dec; 27(4):367-77.