"Pertussis Toxin" is a descriptor in the National Library of Medicine's controlled vocabulary thesaurus,
MeSH (Medical Subject Headings). Descriptors are arranged in a hierarchical structure,
which enables searching at various levels of specificity.
One of the virulence factors produced by BORDETELLA PERTUSSIS. It is a multimeric protein composed of five subunits S1 - S5. S1 contains mono ADPribose transferase activity.
- Pertussis Toxin
- Toxin, Pertussis
- Lymphocytosis-Promoting Factor
- Lymphocytosis Promoting Factor
- Islet-Activating Protein
- Islet Activating Protein
- Histamine-Sensitizing Factor
- Histamine Sensitizing Factor
- IAP Pertussis Toxin
- Pertussis Toxin, IAP
- Toxin, IAP Pertussis
- Islets-Activating Protein
- Islets Activating Protein
Below are MeSH descriptors whose meaning is more general than "Pertussis Toxin".
Below are MeSH descriptors whose meaning is more specific than "Pertussis Toxin".
This graph shows the total number of publications written about "Pertussis Toxin" by people in this website by year, and whether "Pertussis Toxin" was a major or minor topic of these publications.
To see the data from this visualization as text, click here.
|Year||Major Topic||Minor Topic||Total|
To return to the timeline, click here.
Below are the most recent publications written about "Pertussis Toxin" by people in Profiles.
TRPC6 channel translocation into phagosomal membrane augments phagosomal function. Proc Natl Acad Sci U S A. 2015 Nov 24; 112(47):E6486-95.
Depolarization induces a conformational change in the binding site region of the M2 muscarinic receptor. Proc Natl Acad Sci U S A. 2012 Jan 03; 109(1):285-90.
Lysophosphatidic acid induces interleukin-13 (IL-13) receptor alpha2 expression and inhibits IL-13 signaling in primary human bronchial epithelial cells. J Biol Chem. 2007 Apr 06; 282(14):10172-9.
G protein-coupled endothelial receptor for atypical cannabinoid ligands modulates a Ca2+-dependent K+ current. J Biol Chem. 2003 Nov 14; 278(46):46188-94.
Presentation of antigen by endothelial cells and chemoattraction are required for homing of insulin-specific CD8+ T cells. J Exp Med. 2003 Mar 03; 197(5):643-56.
Critical involvement of p38 MAP kinase in pertussis toxin-induced cytoskeletal reorganization and lung permeability. FASEB J. 2002 Jul; 16(9):1064-76.
Protein kinase A-mediated phosphorylation of the beta 2-adrenergic receptor regulates its coupling to Gs and Gi. Demonstration in a reconstituted system. J Biol Chem. 2002 Aug 23; 277(34):31249-56.
Regulation of eosinophil function by phosphatidylinositol-specific PLC and cytosolic PLA(2). Am J Physiol Lung Cell Mol Physiol. 2001 Oct; 281(4):L844-51.
Role of pertussis toxin-sensitive G-proteins in synaptic transmission and plasticity at corticostriatal synapses. J Neurophysiol. 2000 Jan; 83(1):60-9.
Coupling of beta2-adrenoceptor to Gi proteins and its physiological relevance in murine cardiac myocytes. Circ Res. 1999 Jan 8-22; 84(1):43-52.