Vacuolar Proton-Translocating ATPases
"Vacuolar Proton-Translocating ATPases" is a descriptor in the National Library of Medicine's controlled vocabulary thesaurus,
MeSH (Medical Subject Headings). Descriptors are arranged in a hierarchical structure,
which enables searching at various levels of specificity.
Proton-translocating ATPases that are involved in acidification of a variety of intracellular compartments.
| Descriptor ID |
D025262
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| MeSH Number(s) |
D08.811.277.040.025.325.875 D08.811.913.696.650.150.500.875 D12.776.157.530.450.250.875.500.875 D12.776.543.585.450.250.875.500.875
|
| Concept/Terms |
Vacuolar Proton-Translocating ATPases- Vacuolar Proton-Translocating ATPases
- Lysosomal Proton-Translocating ATPases
- ATPases, Lysosomal Proton-Translocating
- Lysosomal Proton Translocating ATPases
- Proton-Translocating ATPases, Lysosomal
- V-Type ATPase
- ATPase, V-Type
- V Type ATPase
- Vacuolar ATPase
- ATPase, Vacuolar
- Vacuolar F(1)F(0) ATPases
- Vacuolar H+-ATPase
- H+-ATPase, Vacuolar
- Vacuolar H+ ATPase
- Vacuolar Membrane H(+)-ATPase
- Lysosomal F(1)F(0) ATPase
- Vacuolar F(1)F(0) ATPase
|
Below are MeSH descriptors whose meaning is more general than "Vacuolar Proton-Translocating ATPases".
Below are MeSH descriptors whose meaning is more specific than "Vacuolar Proton-Translocating ATPases".
This graph shows the total number of publications written about "Vacuolar Proton-Translocating ATPases" by people in this website by year, and whether "Vacuolar Proton-Translocating ATPases" was a major or minor topic of these publications.
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| Year | Major Topic | Minor Topic | Total |
|---|
| 1994 | 0 | 1 | 1 |
| 1997 | 1 | 0 | 1 |
| 1998 | 1 | 0 | 1 |
| 2002 | 1 | 0 | 1 |
| 2005 | 1 | 0 | 1 |
| 2007 | 0 | 1 | 1 |
| 2009 | 0 | 1 | 1 |
| 2012 | 1 | 1 | 2 |
| 2015 | 0 | 1 | 1 |
| 2017 | 0 | 1 | 1 |
| 2018 | 1 | 0 | 1 |
| 2019 | 1 | 0 | 1 |
| 2022 | 1 | 0 | 1 |
| 2023 | 1 | 0 | 1 |
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Below are the most recent publications written about "Vacuolar Proton-Translocating ATPases" by people in Profiles.
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ATP6V0C variants impair V-ATPase function causing a neurodevelopmental disorder often associated with epilepsy. Brain. 2023 04 19; 146(4):1357-1372.
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Phenotypic and genetic spectrum of ATP6V1A encephalopathy: a disorder of lysosomal homeostasis. Brain. 2022 08 27; 145(8):2687-2703.
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Lipid droplet velocity is a microenvironmental sensor of aggressive tumors regulated by V-ATPase and PEDF. Lab Invest. 2019 12; 99(12):1822-1834.
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De novo mutations of the ATP6V1A gene cause developmental encephalopathy with epilepsy. Brain. 2018 06 01; 141(6):1703-1718.
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Reconstructing Ancient Proteins to Understand the Causes of Structure and Function. Annu Rev Biophys. 2017 05 22; 46:247-269.
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Altered autophagic flux enhances inflammatory responses during inflammation-induced preterm labor. Sci Rep. 2015 Mar 23; 5:9410.
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A role for presenilins in autophagy revisited: normal acidification of lysosomes in cells lacking PSEN1 and PSEN2. J Neurosci. 2012 Jun 20; 32(25):8633-48.
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Evolution of increased complexity in a molecular machine. Nature. 2012 Jan 09; 481(7381):360-4.
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Novel loci for major depression identified by genome-wide association study of Sequenced Treatment Alternatives to Relieve Depression and meta-analysis of three studies. Mol Psychiatry. 2011 Feb; 16(2):202-15.
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Inhibition of renin: an updated review of the development of renin inhibitors. Curr Opin Investig Drugs. 2007 Sep; 8(9):750-7.