"Myocardial Reperfusion" is a descriptor in the National Library of Medicine's controlled vocabulary thesaurus,
MeSH (Medical Subject Headings). Descriptors are arranged in a hierarchical structure,
which enables searching at various levels of specificity.
Generally, restoration of blood supply to heart tissue which is ischemic due to decrease in normal blood supply. The decrease may result from any source including atherosclerotic obstruction, narrowing of the artery, or surgical clamping. Reperfusion can be induced to treat ischemia. Methods include chemical dissolution of an occluding thrombus, administration of vasodilator drugs, angioplasty, catheterization, and artery bypass graft surgery. However, it is thought that reperfusion can itself further damage the ischemic tissue, causing MYOCARDIAL REPERFUSION INJURY.
| Descriptor ID |
D015425
|
| MeSH Number(s) |
E04.100.700.600 E05.680.730.600
|
| Concept/Terms |
Myocardial Reperfusion- Myocardial Reperfusion
- Reperfusion, Myocardial
- Myocardial Reperfusions
- Reperfusions, Myocardial
- Coronary Reperfusion
- Coronary Reperfusions
- Reperfusion, Coronary
- Reperfusions, Coronary
|
Below are MeSH descriptors whose meaning is more general than "Myocardial Reperfusion".
Below are MeSH descriptors whose meaning is more specific than "Myocardial Reperfusion".
This graph shows the total number of publications written about "Myocardial Reperfusion" by people in this website by year, and whether "Myocardial Reperfusion" was a major or minor topic of these publications.
To see the data from this visualization as text,
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| Year | Major Topic | Minor Topic | Total |
|---|
| 1998 | 1 | 0 | 1 |
| 1999 | 0 | 1 | 1 |
| 2000 | 0 | 2 | 2 |
| 2001 | 0 | 1 | 1 |
| 2002 | 1 | 1 | 2 |
| 2003 | 1 | 1 | 2 |
| 2004 | 0 | 1 | 1 |
| 2009 | 0 | 1 | 1 |
| 2012 | 0 | 1 | 1 |
| 2021 | 0 | 1 | 1 |
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Below are the most recent publications written about "Myocardial Reperfusion" by people in Profiles.
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Rapid postmortem ventilation improves donor lung viability by extending the tolerable warm ischemic time after cardiac death in mice. Am J Physiol Lung Cell Mol Physiol. 2021 10 01; 321(4):L653-L662.
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ACCF/AHA/SCAI 2013 update of the clinical competence statement on coronary artery interventional procedures: a report of the American College of Cardiology Foundation/American Heart Association/American College of Physicians Task Force on Clinical Competence and Training (writing committee to revise the 2007 clinical competence statement on cardiac interventional procedures). Circulation. 2013 Jul 23; 128(4):436-72.
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Acute myocardial infarction. Dis Mon. 2013 Mar; 59(3):83-96.
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In vivo, cardiac-specific knockdown of a target protein, malic enzyme-1, in rat via adenoviral delivery of DNA for non-native miRNA. Curr Gene Ther. 2012 Dec; 12(6):454-62.
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Accuracy of combined endocardial and epicardial electroanatomic mapping of a reperfused porcine infarct model: a comparison of electrofield and magnetic systems with histopathologic correlation. Heart Rhythm. 2011 Mar; 8(3):439-47.
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Suppressing apoptosis with milrinone simulating extracorporeal circulation: a pilot study. Thorac Cardiovasc Surg. 2010 Aug; 58(5):285-90.
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The nonselective beta-blocker carvedilol suppresses apoptosis in human cardiac tissue: a pilot study. Heart Surg Forum. 2010 Aug; 13(4):E218-22.
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Autologous mesenchymal stem cells produce reverse remodelling in chronic ischaemic cardiomyopathy. Eur Heart J. 2009 Nov; 30(22):2722-32.
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[ST myocardial infarction with spontaneous coronary reperfusion]. Harefuah. 2006 May; 145(5):367-70, 397, 396.
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Adenoviral-mediated transfer of vascular endothelial growth factor 121 cDNA enhances myocardial perfusion and exercise performance in the nonischemic state. J Thorac Cardiovasc Surg. 2004 Feb; 127(2):535-40.