Receptors, Interleukin-1 Type I
"Receptors, Interleukin-1 Type I" is a descriptor in the National Library of Medicine's controlled vocabulary thesaurus,
MeSH (Medical Subject Headings). Descriptors are arranged in a hierarchical structure,
which enables searching at various levels of specificity.
An interleukin-1 receptor subtype that is involved in signaling cellular responses to INTERLEUKIN-1ALPHA and INTERLEUKIN-1BETA. The binding of this receptor to its ligand causes its favorable interaction with INTERLEUKIN-1 RECEPTOR ACCESSORY PROTEIN and the formation of an activated receptor complex.
| Descriptor ID |
D053573
|
| MeSH Number(s) |
D12.776.543.750.705.852.420.300.500
|
| Concept/Terms |
Receptors, Interleukin-1 Type I- Receptors, Interleukin-1 Type I
- Receptors, Interleukin 1 Type I
- CD121a Antigens
- Receptor Type I, Interleukin-1
- Receptor Type I, Interleukin 1
- Interleukin-1 Receptor Type I
- Interleukin 1 Receptor Type I
- Antigens, CDw121a
- CDw121a Antigens
- Interleukin-1 Receptors Type I
- Interleukin 1 Receptors Type I
|
Below are MeSH descriptors whose meaning is more general than "Receptors, Interleukin-1 Type I".
Below are MeSH descriptors whose meaning is more specific than "Receptors, Interleukin-1 Type I".
This graph shows the total number of publications written about "Receptors, Interleukin-1 Type I" by people in this website by year, and whether "Receptors, Interleukin-1 Type I" was a major or minor topic of these publications.
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| Year | Major Topic | Minor Topic | Total |
|---|
| 2006 | 0 | 1 | 1 |
| 2014 | 0 | 1 | 1 |
| 2018 | 1 | 0 | 1 |
| 2021 | 0 | 1 | 1 |
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Below are the most recent publications written about "Receptors, Interleukin-1 Type I" by people in Profiles.
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NLRP3 Inflammasome Mediates Immune-Stromal Interactions in Vasculitis. Circ Res. 2021 10 15; 129(9):e183-e200.
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A Critical Role of the IL-1ß-IL-1R Signaling Pathway in Skin Inflammation and Psoriasis Pathogenesis. J Invest Dermatol. 2019 01; 139(1):146-156.
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Unexpected role for IL-17 in protective immunity against hypervirulent Mycobacterium tuberculosis HN878 infection. PLoS Pathog. 2014 May; 10(5):e1004099.
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Signaling via the type I IL-1 and TNF receptors is necessary for bacterially induced preterm labor in a murine model. Am J Obstet Gynecol. 2006 May; 194(5):1334-40.